C1q as a unique player in angiogenesis with therapeutic implication in wound healing. - Wikidata - awgldk - TriplyDB

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

http://www.wikidata.org/entity/Q37660089

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Emerging and Novel Functions of Complement Protein C1q Phagocytosis of apoptotic cells in homeostasis The endothelial deprotection hypothesis for lupus pathogenesis: the dual role of C1q as a mediator of clearance and regulator of endothelial permeability Pericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis.Complement deficiency promotes cutaneous wound healing in mice.Prognostic stromal gene signatures in breast cancer.Serological and Genetic Evidence for Altered Complement System Functionality in Systemic Lupus Erythematosus: Findings of the GAPAID Consortium.Treponema pallidum (syphilis) antigen TpF1 induces angiogenesis through the activation of the IL-8 pathway C1q acts in the tumour microenvironment as a cancer-promoting factor independently of complement activation.Tumor-host signaling interaction reveals a systemic, age-dependent splenic immune influence on tumor development.Targeted complement inhibition and microvasculature in transplants: a therapeutic perspective.Humoral innate immunity at the crossroad between microbe and matrix recognition: The role of PTX3 in tissue damage.Innate immune mediators in cancer: between defense and resistance.Endothelial cells: source, barrier, and target of defensive mediators.The complement and contact activation systems: partnership in pathogenesis beyond angioedema.C1ql1/Ctrp14 and C1ql4/Ctrp11 promote angiogenesis of endothelial cells through activation of ERK1/2 signal pathway.C1q: A fresh look upon an old molecule.Cell-specific deletion of C1qa identifies microglia as the dominant source of C1q in mouse brain.The non-inflammatory role of C1q during Her2/neu-driven mammary carcinogenesis.The E3 ligase Itch knockout mice show hyperproliferation and wound healing alteration.Complement Protein C1q Binds to Hyaluronic Acid in the Malignant Pleural Mesothelioma Microenvironment and Promotes Tumor Growth.Interaction of Complement Defence Collagens C1q and Mannose-Binding Lectin with BMP-1/Tolloid-like Proteinases.Hemocompatibility of Ca2+ -Crosslinked Nanocellulose Hydrogels: Toward Efficient Management of Hemostasis.Targeting complement-mediated immunoregulation for cancer immunotherapy.A model to study complement involvement in experimental retinal degeneration.[The complement system: a double edge sword in tumor progression].Complement component C1q as potential diagnostic but not predictive marker of preeclampsia.Cell-autonomous and cell non-autonomous downregulation of tumor suppressor DAB2IP by microRNA-149-3p promotes aggressiveness of cancer cells.

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P2860

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

http://www.wikidata.org/entity/Q37660089

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 March 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@en

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@nl

type

label

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@en

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@nl

prefLabel

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@en

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

@nl

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PNAS.1311968111

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Proceedings of the National Academy of Sciences of the United States of America

P1476

C1q as a unique player in angiogenesis with therapeutic implication in wound healing.

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Berhane Ghebrehiwet

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Carine Munaut

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Carla Guarnotta

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Chiara Agostinis

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Claudio Tripodo

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Fleur Bossi

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Francesco Tedesco

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Giovanni Papa

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Guang Sheng Ling

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Gustavo Baldassarre

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P2860

Cooperation of C1q receptors and integrins in C1q-mediated endothelial cell adhesion and spreading

Drusen complement components C3a and C5a promote choroidal neovascularization

Vascular permeability factor/vascular endothelial growth factor, microvascular hyperpermeability, and angiogenesis

Tumors: wounds that do not heal. Similarities between tumor stroma generation and wound healing

CD59, a complement regulatory protein, controls choroidal neovascularization in a mouse model of wet-type age-related macular degeneration

Role of complement and complement membrane attack complex in laser-induced choroidal neovascularization

Molecular mechanisms and clinical applications of angiogenesis

Angiogenesis in life, disease and medicine

Complement activation via alternative pathway is critical in the development of laser-induced choroidal neovascularization: role of factor B and factor H.

Complement-mediated inhibition of neovascularization reveals a point of convergence between innate immunity and angiogenesis.

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10.1073/PNAS.1311968111

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