Toll-Like Receptor 2 Is Required for Inflammatory Process Development during Leishmania infantum Infection. - Wikidata - awgldk - TriplyDB

Toll-Like Receptor 2 Is Required for Inflammatory Process Development during Leishmania infantum Infection.

Toll-Like Receptor 2 Is Required for Inflammatory Process Development during Leishmania infantum Infection.

http://www.wikidata.org/entity/Q37660791

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The Equivocal Role of Th17 Cells and Neutrophils on Immunopathogenesis of Leishmaniasis.Leishmania infantum Induces the Release of sTREM-1 in Visceral Leishmaniasis.

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Toll-Like Receptor 2 Is Required for Inflammatory Process Development during Leishmania infantum Infection.

http://www.wikidata.org/entity/Q37660791

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article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 23 February 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

@en

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

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type

label

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

@en

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

@nl

prefLabel

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

@en

Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection.

@nl

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FMICB.2017.00262

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Frontiers in Microbiology

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Toll-Like Receptor 2 Is Requir ...... Leishmania infantum Infection

@en

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Jéssica L da Costa

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Laís A Sacramento

http://www.w3.org/2001/XMLSchema#string

Mikhael H F de Lima

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Pedro A Sampaio

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Roque P Almeida

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P2860

In vivo involvement of polymorphonuclear neutrophils in Leishmania infantum infection

The host-protective effect of arabinosylated lipoarabinomannan against Leishmania donovani infection is associated with restoration of IFN-γ responsiveness

iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice

Pathogen recognition and innate immunity

CD8 cells of patients with diffuse cutaneous leishmaniasis display functional exhaustion: the latter is reversed, in vitro, by TLR2 agonists.

Organ-specific immune responses associated with infectious disease.

Developmental modification of lipophosphoglycan during the differentiation of Leishmania major promastigotes to an infectious stage.

Arginine usage in mycobacteria-infected macrophages depends on autocrine-paracrine cytokine signaling

Unleashing the potential of NOD- and Toll-like agonists as vaccine adjuvants.

Glycoinositolphospholipids from Leishmania braziliensis and L. infantum: modulation of innate immune system and variations in carbohydrate structure

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262

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scholarly article

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10.3389/FMICB.2017.00262

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P478

8

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Fernando Queiroz Cunha

Vanessa Carregaro

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2017-02-23T00:00:00Z

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28280488

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Toll-like receptor

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5322192

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