Ischemic neurons activate astrocytes to disrupt endothelial barrier via increasing VEGF expression.
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Therapy Effects of Bone Marrow Stromal Cells on Ischemic StrokeGrowth factors for the treatment of ischemic brain injury (growth factor treatment)Hemorrhagic Transformation after Tissue Plasminogen Activator Reperfusion Therapy for Ischemic Stroke: Mechanisms, Models, and Biomarkers.Pericytes contribute to the disruption of the cerebral endothelial barrier via increasing VEGF expression: implications for stroke.Cellular connections, microenvironment and brain angiogenesis in diabetes: Lost communication signals in the post-stroke period.Re-exposure to the hypobaric hypoxic brain injury of high altitude: plasma S100B levels and the possible effect of acclimatisation on blood-brain barrier dysfunction.Mismatch of Low Perfusion and High Permeability Predicts Hemorrhagic Transformation Region in Acute Ischemic Stroke Patients Treated with Intra-arterial ThrombolysisOne-Compound-Multi-Target: Combination Prospect of Natural Compounds with Thrombolytic Therapy in Acute Ischemic Stroke.Pathogenesis of brain edema and investigation into anti-edema drugs.Blood-brain barrier breakdown and neovascularization processes after stroke and traumatic brain injury.Disruption of in vitro endothelial barrier integrity by Japanese encephalitis virus-Infected astrocytes.β2-Adrenergic Receptor-Mediated HIF-1α Upregulation Mediates Blood Brain Barrier Damage in Acute Cerebral Ischemia.Small-animal PET demonstrates brain metabolic change after using bevacizumab in a rat model of cerebral ischemic injury.Influence of cortical synaptic input on striatal neuronal dendritic arborization and sensitivity to excitotoxicity in corticostriatal coculture.Vulnerability of primary neurons derived from Tg2576 Alzheimer mice to oxygen and glucose deprivation: role of intraneuronal amyloid-β accumulation and astrocytes.Blood-brain barrier dysfunction and recovery after ischemic stroke.BIRC5 is a novel target of peroxisome proliferator‑activated receptor γ in brain microvascular endothelium cells during cerebral ischemia.Astrocyte-derived lipocalin-2 mediates hippocampal damage and cognitive deficits in experimental models of vascular dementia.Astrocytes spatially restrict VEGF signaling by polarized secretion and incorporation of VEGF into the actively assembling extracellular matrix.Lack of NG2 exacerbates neurological outcome and modulates glial responses after traumatic brain injury.Brain vascular heterogeneity: implications for disease pathogenesis and design of in vitro blood-brain barrier models.The Janus Face of VEGF in Stroke.Inhibition of HIF-1α Reduced Blood Brain Barrier Damage by Regulating MMP-2 and VEGF During Acute Cerebral Ischemia
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P2860
Ischemic neurons activate astrocytes to disrupt endothelial barrier via increasing VEGF expression.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 06 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@en
Ischemic neurons activate astr ...... ia increasing VEGF expression.
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type
label
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@en
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@nl
prefLabel
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@en
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@nl
P2093
P2860
P356
P1476
Ischemic neurons activate astr ...... ia increasing VEGF expression.
@en
P2093
Ke Jian Liu
Wei-Lin Yang
Wenlan Liu
Xu-Jun Qin
Ying-Na Li
Zhifeng Qi
P2860
P304
P356
10.1111/JNC.12611
P407
P577
2013-12-06T00:00:00Z