Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD. - Wikidata - awgldk - TriplyDB

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

http://www.wikidata.org/entity/Q37670317

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Impaired integrin β3 delays endothelial cell regeneration and contributes to arteriovenous graft failure in mice.In vivo nanoparticle assessment of pathological endothelium predicts the development of inflow stenosis in murine arteriovenous fistula.Protective role of insulin-like growth factor-1 receptor in endothelial cells against unilateral ureteral obstruction-induced renal fibrosis.FSP-1 Impairs the Function of Endothelium Leading to Failure of Arteriovenous Grafts in Diabetic Mice Prevention of Venous Neointimal Hyperplasia by a Multitarget Receptor Tyrosine Kinase Inhibitor.A new model of an arteriovenous fistula in chronic kidney disease in the mouse: beneficial effects of upregulated heme oxygenase-1 Hemodynamic Shear Stress and Endothelial Dysfunction in Hemodialysis Access.Notch signal integration in the vasculature during remodeling Notch Signaling in Endothelial Cells: Is It the Therapeutic Target for Vascular Neointimal Hyperplasia?New Insights into Dialysis Vascular Access: Molecular Targets in Arteriovenous Fistula and Arteriovenous Graft Failure and Their Potential to Improve Vascular Access Outcomes.Notch ties a knot on fistula maturation.Migration of smooth muscle cells from the arterial anastomosis of arteriovenous fistulas requires Notch activation to form neointima.Reduced Expression of Glutathione S-Transferase α 4 Promotes Vascular Neointimal Hyperplasia in CKD.Interleukin-9 mediates chronic kidney disease-dependent vein graft disease: a role for mast cells.Yap/Taz Deletion in Gli+ Cell-Derived Myofibroblasts Attenuates Fibrosis.Long Noncoding RNA uc001pwg.1 Is Downregulated in Neointima in Arteriovenous Fistulas and Mediates the Function of Endothelial Cells Derived from Pluripotent Stem Cells.Jagged1/Notch2 controls kidney fibrosis via Tfam-mediated metabolic reprogramming

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Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

http://www.wikidata.org/entity/Q37670317

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 30 January 2014

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@en

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@nl

type

label

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@en

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@nl

prefLabel

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@en

Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@nl

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Journal of the American Society of Nephrology

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Blocking Notch in endothelial cells prevents arteriovenous fistula failure despite CKD.

@en

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Anlin Liang

http://www.w3.org/2001/XMLSchema#string

Guofeng Han

http://www.w3.org/2001/XMLSchema#string

Jinlong Luo

http://www.w3.org/2001/XMLSchema#string

Jizhong Cheng

http://www.w3.org/2001/XMLSchema#string

Ming Liang

http://www.w3.org/2001/XMLSchema#string

William E Mitch

http://www.w3.org/2001/XMLSchema#string

Yun Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Integration of a Notch-dependent mesenchymal gene program and Bmp2-driven cell invasiveness regulates murine cardiac valve formation.

Essential role of endothelial Notch1 in angiogenesis

Endothelial Notch4 signaling induces hallmarks of brain arteriovenous malformations in mice.

Notch1 activation in mice causes arteriovenous malformations phenocopied by ephrinB2 and EphB4 mutants.

Heart valve development: endothelial cell signaling and differentiation

Satellite cell dysfunction and impaired IGF-1 signaling cause CKD-induced muscle atrophy

Notch activation results in phenotypic and functional changes consistent with endothelial-to-mesenchymal transformation.

MCP-1 contributes to arteriovenous fistula failure.

Pharmacological inhibition of myostatin suppresses systemic inflammation and muscle atrophy in mice with chronic kidney disease.

Increasing arteriovenous fistulas in hemodialysis patients: problems and solutions.

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773-783

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scholarly article

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10.1681/ASN.2013050490

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4

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25

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2014-01-30T00:00:00Z

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24480830

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3968498

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