Epigenetic modification restores functional PR expression in endometrial cancer cells. - Wikidata - awgldk - TriplyDB

Epigenetic modification restores functional PR expression in endometrial cancer cells.

Epigenetic modification restores functional PR expression in endometrial cancer cells.

http://www.wikidata.org/entity/Q37671692

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Past, present, and future of hormonal therapy in recurrent endometrial cancer Role of nuclear progesterone receptor isoforms in uterine pathophysiology.Systematic dissection of the mechanisms underlying progesterone receptor downregulation in endometrial cancer Progesterone action in breast, uterine, and ovarian cancers.Inverse Relationship between Progesterone Receptor and Myc in Endometrial Cancer.Progesterone receptor isoforms A and B: new insights into the mechanism of progesterone resistance for the treatment of endometrial carcinoma.Nucleophosmin/B23 is a negative regulator of estrogen receptor α expression via AP2γ in endometrial cancer cells.Moving forward with actionable therapeutic targets and opportunities in endometrial cancer: NCI clinical trials planning meeting report on identifying key genes and molecular pathways for targeted endometrial cancer trials.Panobinostat Enhances Growth Suppressive Effects of Progestin on Endometrial Carcinoma by Increasing Progesterone Receptor and Mitogen-Inducible Gene-6.

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Epigenetic modification restores functional PR expression in endometrial cancer cells.

http://www.wikidata.org/entity/Q37671692

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on January 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@en

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@nl

type

label

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@en

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@nl

prefLabel

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@en

Epigenetic modification restores functional PR expression in endometrial cancer cells.

@nl

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Current Pharmaceutical Design

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Epigenetic modification restores functional PR expression in endometrial cancer cells.

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P2093

Christopher J Winters

http://www.w3.org/2001/XMLSchema#string

Eric J Devor

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Kimberly K Leslie

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Kristina W Thiel

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Shujie Yang

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Xiangbing Meng

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Xiaoyue Liu

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Xinjun Wang

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Xue Xiao

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Yichen Jia

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P2860

Chemoproteomics profiling of HDAC inhibitors reveals selective targeting of HDAC complexes

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Progesterone receptor B gene inactivation and CpG hypermethylation in human uterine endometrial cancer.

Progesterone: the ultimate endometrial tumor suppressor.

Knockdown of MTDH sensitizes endometrial cancer cells to cell death induction by death receptor ligand TRAIL and HDAC inhibitor LBH589 co-treatment.

Phosphorylation of human progesterone receptors at serine-294 by mitogen-activated protein kinase signals their degradation by the 26S proteasome.

Progesterone receptor isoform identification and subcellular localization in endometrial cancer

Endometrial cancer: reviving progesterone therapy in the molecular age.

Down-regulation of the progesterone receptor by the methylation of progesterone receptor gene in endometrial cancer cells.

Epigenetic-mediated upregulation of progesterone receptor B gene in endometrial cancer cell lines.

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1874-1880

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scholarly article

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10.2174/13816128113199990532

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11

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2014-01-01T00:00:00Z

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