Electrophysiology and metabolism of caveolin-3-overexpressing mice. - Wikidata - awgldk - TriplyDB

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

http://www.wikidata.org/entity/Q37679792

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Modulation of caveolins, integrins and plasma membrane repair proteins in anthracycline-induced heart failure in rabbits.Potential Roles of Serum Caveolin-3 Levels in Patients with Atrial Fibrillation.Cardiac striatin interacts with caveolin-3 and calmodulin in a calcium sensitive manner and regulates cardiomyocyte spontaneous contraction rate.Caveolin-1/-3: therapeutic targets for myocardial ischemia/reperfusion injury.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Species-Dependent Mechanisms of Cardiac Arrhythmia: A Cellular Focus.Cavin-1 deficiency modifies myocardial and coronary function, stretch responses and ischaemic tolerance: roles of NOS over-activity.Cardioprotective kinase signaling to subsarcolemmal and interfibrillar mitochondria is mediated by caveolar structures.Caveolin-1 facilitated KCNA5 expression, promoting breast cancer viability

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Electrophysiology and metabolism of caveolin-3-overexpressing mice.

http://www.wikidata.org/entity/Q37679792

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 March 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@en

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@nl

type

label

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@en

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@nl

prefLabel

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@en

Electrophysiology and metabolism of caveolin-3-overexpressing mice.

@nl

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S00395-016-0542-9

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Basic Research in Cardiology

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Electrophysiology and metabolism of caveolin-3-overexpressing mice.

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Alice E Zemljic-Harpf

http://www.w3.org/2001/XMLSchema#string

Andrew D McCulloch

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David M Roth

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Hemal H Patel

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Jan M Schilling

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Judith K Yu

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Kevin P Vincent

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Leonid Tyan

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Ravi C Balijepalli

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Yousuke T Horikawa

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P2860

Basic Mechanisms of Cardiac Impulse Propagation and Associated Arrhythmias

Novel mechanism for sudden infant death syndrome: persistent late sodium current secondary to mutations in caveolin-3

Caveolin-3 regulates protein kinase A modulation of the Ca(V)3.2 (alpha1H) T-type Ca2+ channels

Mutant caveolin-3 induces persistent late sodium current and is associated with long-QT syndrome

SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome

Cardiac-specific overexpression of caveolin-3 induces endogenous cardiac protection by mimicking ischemic preconditioning

Cardiac-specific overexpression of caveolin-3 attenuates cardiac hypertrophy and increases natriuretic peptide expression and signaling

Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesis

Interaction of risk factors, comorbidities, and comedications with ischemia/reperfusion injury and cardioprotection by preconditioning, postconditioning, and remote conditioning

Molecular basis of cardioprotection: signal transduction in ischemic pre-, post-, and remote conditioning

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s00395-016-0542-9

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10.1007/S00395-016-0542-9

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3

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2016-03-29T00:00:00Z

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electrophysiology

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