Cav1.2 channels mediate persistent chronic stress-induced behavioral deficits that are associated with prefrontal cortex activation of the p25/Cdk5-glucocorticoid receptor pathway.
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From Gene to Behavior: L-Type Calcium Channel Mechanisms Underlying Neuropsychiatric Symptoms.Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood.Proteolytic processing of the L-type Ca 2+ channel alpha 11.2 subunit in neurons.Extinction of Contextual Cocaine Memories Requires Cav1.2 within D1R-Expressing Cells and Recruits Hippocampal Cav1.2-Dependent Signaling Mechanisms.Comprehensive analysis of two Shank3 and the Cacna1c mouse models of autism spectrum disorder.Downregulation of the psychiatric susceptibility gene Cacna1c promotes mitochondrial resilience to oxidative stress in neuronal cells.
P2860
Cav1.2 channels mediate persistent chronic stress-induced behavioral deficits that are associated with prefrontal cortex activation of the p25/Cdk5-glucocorticoid receptor pathway.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 February 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
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Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
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type
label
Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
@en
Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
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prefLabel
Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
@en
Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
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P2093
P2860
P1476
Cav1.2 channels mediate persis ...... ucocorticoid receptor pathway.
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P2093
Anjali M Rajadhyaksha
Bryant K Rizzo
Charlotte C Bavley
P2860
P356
10.1016/J.YNSTR.2017.02.004
P577
2017-02-24T00:00:00Z