IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections. - Wikidata - awgldk - TriplyDB

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

http://www.wikidata.org/entity/Q37693697

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Host Iron Nutritional Immunity Induced by a Live Yersinia pestis Vaccine Strain Is Associated with Immediate Protection against Plague.Gut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease.IL-22: Scavenging beyond the barrier.An Organismal Model for Gene Regulatory Networks in the Gut-Associated Immune Response.Antimicrobial IL-22.Excessive Iron Availability Caused by Disorders of Interleukin-10 and Interleukin-22 Contributes to High Altitude Polycythemia.The Interleukin-20 Cytokines in Intestinal Diseases.Modulation of Iron Metabolism in Response to Infection: Twists for All Tastes Cross-Talk Between Iron and Glucose Metabolism in the Establishment of Disease Tolerance

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IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

http://www.wikidata.org/entity/Q37693697

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 February 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@en

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@nl

type

label

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@en

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@nl

prefLabel

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@en

IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@nl

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SCIIMMUNOL.AAI8371

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Science immunology

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IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.

@en

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Gabriel Núñez

http://www.w3.org/2001/XMLSchema#string

Gustavo Caballero-Flores

http://www.w3.org/2001/XMLSchema#string

Hideki Hara

http://www.w3.org/2001/XMLSchema#string

José L Puente

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Kei Sakamoto

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Naohiro Inohara

http://www.w3.org/2001/XMLSchema#string

Nobuhiko Kamada

http://www.w3.org/2001/XMLSchema#string

Yun-Gi Kim

http://www.w3.org/2001/XMLSchema#string

P2860

Interleukin (IL)-22, a novel human cytokine that signals through the interferon receptor-related proteins CRF2-4 and IL-22R

A bacterial effector targets host DH-PH domain RhoGEFs and antagonizes macrophage phagocytosis

Identification and functional characterization of a second chain of the interleukin-10 receptor complex.

Interleukin-22 Signaling in the Regulation of Intestinal Health and Disease

Disease tolerance as a defense strategy

Population-based epidemiology and microbiology of community-onset bloodstream infections

Lack of Plasma Protein Hemopexin Results in Increased Duodenal Iron Uptake

Iron homeostasis in host defence and inflammation

An IL-23R/IL-22 Circuit Regulates Epithelial Serum Amyloid A to Promote Local Effector Th17 Responses

IL-22 increases the innate immunity of tissues

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scholarly article

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10.1126/SCIIMMUNOL.AAI8371

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8

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2

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Emanuela Tolosano

Miguel P. Soares

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2017-02-03T00:00:00Z

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28286877

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5345941

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