Potentiation of colon cancer susceptibility in mice by colonic epithelial PPAR-δ/β overexpression. - Wikidata - awgldk - TriplyDB

Potentiation of colon cancer susceptibility in mice by colonic epithelial PPAR-δ/β overexpression.

Potentiation of colon cancer susceptibility in mice by colonic epithelial PPAR-δ/β overexpression.

http://www.wikidata.org/entity/Q37696705

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15-Lipoxygenase-1 suppression of colitis-associated colon cancer through inhibition of the IL-6/STAT3 signaling pathway.PPARδ and PGE2 signaling pathways communicate and connect inflammation to colorectal cancer Establishing the Role of PPARβ/δ in Carcinogenesis Systemic Chromosome Instability Resulted in Colonic Transcriptomic Changes in Metabolic, Proliferation, and Stem Cell Regulators in Sgo1-/+ Mice Metastasis regulation by PPARD expression in cancer cells.ALOX15 as a suppressor of inflammation and cancer: Lost in the link.Therapeutic potential of GW501516 and the role of Peroxisome proliferator-activated receptor β/δ and B-cell lymphoma 6 in inflammatory signaling in human pancreatic cancer cells.Peroxisome proliferator-activated receptors (PPARs) are potential drug targets for cancer therapy ROS release by PPARβ/δ-null fibroblasts reduces tumor load through epithelial antioxidant response.The Anticancer Potential of Peroxisome Proliferator-Activated Receptor Antagonists.PPAR-delta promotes survival of chronic lymphocytic leukemia cells in energetically unfavorable conditions.Differential DNA methylation profiles of human B lymphocytes and Epstein-Barr virus-immortalized B lymphocytes.Induction of apoptosis in prostate cancer by ginsenoside Rh2.

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Potentiation of colon cancer susceptibility in mice by colonic epithelial PPAR-δ/β overexpression.

http://www.wikidata.org/entity/Q37696705

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 March 2014

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

@en

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

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type

label

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

@en

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

@nl

prefLabel

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

@en

Potentiation of colon cancer s ...... elial PPAR-δ/β overexpression.

@nl

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Journal of the National Cancer Institute

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Potentiation of colon cancer s ...... helial PPAR-δ/β overexpression

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Jeffrey S Morris

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Jiang Yu

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Micheline J Moussalli

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Mihai Gagea

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Min Xu

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Russell R Broaddus

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Shoudan Liang

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Sun Il Lee

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Xiangsheng Zuo

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Yuanqing Wu

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The peroxisome proliferator-activated receptor delta, an integrator of transcriptional repression and nuclear receptor signaling.

The role of peroxisome proliferator-activated receptors in carcinogenesis and chemoprevention

Expression of PPARdelta in multistage carcinogenesis of the colorectum: implications of malignant cancer morphology

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Effects of gut-targeted 15-LOX-1 transgene expression on colonic tumorigenesis in mice

PPARdelta is an APC-regulated target of nonsteroidal anti-inflammatory drugs

Differentiation of trophoblast giant cells and their metabolic functions are dependent on peroxisome proliferator-activated receptor beta/delta

Effects of peroxisome proliferator-activated receptor delta on placentation, adiposity, and colorectal cancer.

Estimating the occurrence of false positives and false negatives in microarray studies by approximating and partitioning the empirical distribution of p-values.

Epigenetic identification of ADAMTS18 as a novel 16q23.1 tumor suppressor frequently silenced in esophageal, nasopharyngeal and multiple other carcinomas.

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10.1093/JNCI/DJU052

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Ganiraju Manyam

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2014-03-28T00:00:00Z

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