Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost. - Wikidata - awgldk - TriplyDB

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

http://www.wikidata.org/entity/Q37698142

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Mechanisms of sodium channel clustering and its influence on axonal impulse conduction The olfactomedin domain from gliomedin is a β-propeller with unique structural properties.Postnatal Loss of Neuronal and Glial Neurofascins Differentially Affects Node of Ranvier Maintenance and Myelinated Axon Function.Long-term maintenance of Na+ channels at nodes of Ranvier depends on glial contact mediated by gliomedin and NrCAM.The node of Ranvier in CNS pathology Autoimmune antigenic targets at the node of Ranvier in demyelinating disorders.Subcellular patterning: axonal domains with specialized structure and function.The Nodes of Ranvier: Molecular Assembly and Maintenance.The Axonal Cytoskeleton and the Assembly of Nodes of Ranvier.Chronic peripheral nerve compression disrupts paranodal axoglial junctions.Peroxisomal dysfunctions cause lysosomal storage and axonal Kv1 channel redistribution in peripheral neuropathy.Diagnostic Utility of Auto Antibodies in Inflammatory Nerve Disorders.The paranodal cytoskeleton clusters Na+ channels at nodes of Ranvier.Glial M6B stabilizes the axonal membrane at peripheral nodes of Ranvier.Simultaneous Ablation of Neuronal Neurofascin and Ankyrin G in Young and Adult Mice Reveals Age-Dependent Increase in Nodal Stability in Myelinated Axons and Differential Effects on the Lifespan.

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P2860

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

http://www.wikidata.org/entity/Q37698142

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on April 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@en

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@nl

type

label

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@en

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@nl

prefLabel

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@en

Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@nl

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JNEUROSCI.4662-13.2014

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Journal of Neuroscience

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Differential stability of PNS and CNS nodal complexes when neuronal neurofascin is lost.

@en

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Ao Zhang

http://www.w3.org/2001/XMLSchema#string

Barbara Zonta

http://www.w3.org/2001/XMLSchema#string

Diane L Sherman

http://www.w3.org/2001/XMLSchema#string

Lai-Man N Wu

http://www.w3.org/2001/XMLSchema#string

Peter J Brophy

http://www.w3.org/2001/XMLSchema#string

P2860

Neurofascin is a glial receptor for the paranodin/Caspr-contactin axonal complex at the axoglial junction

Mice lacking sodium channel beta1 subunits display defects in neuronal excitability, sodium channel expression, and nodal architecture

A glial signal consisting of gliomedin and NrCAM clusters axonal Na+ channels during the formation of nodes of Ranvier

Neurofascins are required to establish axonal domains for saltatory conduction

Molecular composition of the node of Ranvier: identification of ankyrin-binding cell adhesion molecules neurofascin (mucin+/third FNIII domain-) and NrCAM at nodal axon segments

Paranodin, a glycoprotein of neuronal paranodal membranes

Axon-glia interactions and the domain organization of myelinated axons requires neurexin IV/Caspr/Paranodin.

Spatiotemporal ablation of myelinating glia-specific neurofascin (Nfasc NF155) in mice reveals gradual loss of paranodal axoglial junctions and concomitant disorganization of axonal domains.

Developing nodes of Ranvier are defined by ankyrin-G clustering and are independent of paranodal axoglial adhesion.

Restricted growth of Schwann cells lacking Cajal bands slows conduction in myelinated nerves.

P304

5083-5088

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scholarly article

P356

10.1523/JNEUROSCI.4662-13.2014

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15

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34

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Anne Desmazieres

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2014-04-01T00:00:00Z

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P698

24719087

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P932

3983793

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