Infection and HLA-G molecules in nasal polyposis. - Wikidata - awgldk - TriplyDB

Infection and HLA-G molecules in nasal polyposis.

Infection and HLA-G molecules in nasal polyposis.

http://www.wikidata.org/entity/Q37704585

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HLA-G Molecules in Autoimmune Diseases and Infections Human Papillomavirus (HPV) Prevalence in Nasal and Antrochoanal Polyps and Association with Clinical Data.Pseudomonas aeruginosa Quorum Sensing Molecule N-(3-Oxododecanoyl)-L-Homoserine-Lactone Induces HLA-G Expression in Human Immune Cells

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Infection and HLA-G molecules in nasal polyposis.

http://www.wikidata.org/entity/Q37704585

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 March 2014

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vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

Infection and HLA-G molecules in nasal polyposis.

@en

Infection and HLA-G molecules in nasal polyposis.

@nl

type

label

Infection and HLA-G molecules in nasal polyposis.

@en

Infection and HLA-G molecules in nasal polyposis.

@nl

prefLabel

Infection and HLA-G molecules in nasal polyposis.

@en

Infection and HLA-G molecules in nasal polyposis.

@nl

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Journal of Immunology Research

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Infection and HLA-G molecules in nasal polyposis

@en

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Antonella Rotola

http://www.w3.org/2001/XMLSchema#string

Antonio Pastore

http://www.w3.org/2001/XMLSchema#string

Claudia Aimoni

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Enrico Fainardi

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Nicola Malagutti

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Roberta Rizzo

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Stefano Pelucchi

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Teresa Pezzolo

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P2860

Differentiation of type 1 T regulatory cells (Tr1) by tolerogenic DC-10 requires the IL-10-dependent ILT4/HLA-G pathway

Human inhibitory receptors Ig-like transcript 2 (ILT2) and ILT4 compete with CD8 for MHC class I binding and bind preferentially to HLA-G

HLA-G up-regulates ILT2, ILT3, ILT4, and KIR2DL4 in antigen presenting cells, NK cells, and T cells

Failure of gamma interferon but not interleukin-10 expression in response to human papillomavirus type 11 E6 protein in respiratory papillomatosis.

Erythroblasts secrete the nonclassical HLA-G molecule from primitive to definitive hematopoiesis.

The PDZ ligand domain of the human papillomavirus type 16 E6 protein is required for E6's induction of epithelial hyperplasia in vivo.

Mechanisms of genomic instability in human cancer: insights from studies with human papillomavirus oncoproteins.

Alternative splicing of HLA-G transcripts yields proteins with primary structures resembling both class I and class II antigens.

Latent papillomavirus infections and their regulation.

Human leukocyte antigen-G is closely associated with tumor immune escape in gastric cancer by increasing local regulatory T cells.

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407430

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scholarly article

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10.1155/2014/407430

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2014

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Valentina Gentili

Daria Bortolotti

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2014-03-06T00:00:00Z

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261754223

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