Neither absence nor excess of FGF23 disturbs murine fetal-placental phosphorus homeostasis or prenatal skeletal development and mineralization.
about
The G protein α subunit variant XLαs promotes inositol 1,4,5-trisphosphate signaling and mediates the renal actions of parathyroid hormone in vivo.Current concepts in perinatal mineral metabolismBone development and mineral homeostasis in the fetus and neonate: roles of the calciotropic and phosphotropic hormones.Maternal Mineral and Bone Metabolism During Pregnancy, Lactation, and Post-Weaning Recovery.FGF23 Is Not Required to Regulate Fetal Phosphorus Metabolism but Exerts Effects Within 12 Hours After Birth.Intestinal Depletion of NaPi-IIb/Slc34a2 in Mice: Renal and Hormonal Adaptation.Placental Vascular Calcification and Cardiovascular Health: It Is Time to Determine How Much of Maternal and Offspring Health Is Written in Stone
P2860
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P2860
Neither absence nor excess of FGF23 disturbs murine fetal-placental phosphorus homeostasis or prenatal skeletal development and mineralization.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 06 March 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Neither absence nor excess of ...... evelopment and mineralization.
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Neither absence nor excess of ...... evelopment and mineralization.
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Neither absence nor excess of ...... evelopment and mineralization.
@en
Neither absence nor excess of ...... evelopment and mineralization.
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prefLabel
Neither absence nor excess of ...... evelopment and mineralization.
@en
Neither absence nor excess of ...... evelopment and mineralization.
@nl
P2093
P2860
P356
P1433
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Neither absence nor excess of ...... development and mineralization
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P2093
Andrew C Karaplis
Beate Lanske
Beth J Kirby
Manoharee Samaraweera
Sandra Cooke-Hubley
P2860
P304
P356
10.1210/EN.2013-2061
P407
P577
2014-03-06T00:00:00Z