Progressive activation of T(H)2/T(H)22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis. - Wikidata - awgldk - TriplyDB

Progressive activation of T(H)2/T(H)22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis.

Progressive activation of T(H)2/T(H)22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis.

http://www.wikidata.org/entity/Q37709681

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Molecular Mechanisms of Cutaneous Inflammatory Disorder: Atopic Dermatitis Sézary Syndrome and Atopic Dermatitis: Comparison of Immunological Aspects and Targets Regulation of T Cell Immunity in Atopic Dermatitis by Microbes: The Yin and Yang of Cutaneous Inflammation Immune Pathways in Atopic Dermatitis, and Definition of Biomarkers through Broad and Targeted Therapeutics Biological Treatments in Atopic Dermatitis Regulation of Dendritic Cell Function in Inflammation New insights into T cells and their signature cytokines in atopic dermatitis Deciphering the complexities of atopic dermatitis: shifting paradigms in treatment approaches The ABC's of managing patients with severe atopic dermatitis Th22 cells in allergic disease The Pathogenetic Effect of Natural and Bacterial Toxins on Atopic Dermatitis Multiple Transcriptome Data Analysis Reveals Biologically Relevant Atopic Dermatitis Signature Genes and Pathways Environmental risk factors and their role in the management of atopic dermatitis.TWEAK mediates inflammation in experimental atopic dermatitis and psoriasis.Upregulation of calprotectin in mild IgE-mediated ovalbumin hypersensitivity.Th2 cytokines increase Staphylococcus aureus alpha toxin-induced keratinocyte death through the signal transducer and activator of transcription 6 (STAT6)Cyclosporine in patients with atopic dermatitis modulates activated inflammatory pathways and reverses epidermal pathology.Staphylococcal and streptococcal superantigen exotoxins Therapeutic strategies in extrinsic atopic dermatitis: focus on inhibition of IL-4 as a new pharmacological approach.Target-oriented therapy: Emerging drugs for atopic dermatitis.The Atopic March: Progression from Atopic Dermatitis to Allergic Rhinitis and Asthma.Staphylococcus aureus alpha toxin suppresses effective innate and adaptive immune responses in a murine dermonecrosis model.Dysbiosis and Staphylococcus aureus Colonization Drives Inflammation in Atopic Dermatitis.Identification of novel immune and barrier genes in atopic dermatitis by means of laser capture microdissection.Meta-analysis derived atopic dermatitis (MADAD) transcriptome defines a robust AD signature highlighting the involvement of atherosclerosis and lipid metabolism pathways Effects of the Staphylococcus aureus and Staphylococcus epidermidis Secretomes Isolated from the Skin Microbiota of Atopic Children on CD4+ T Cell Activation.The role of innate immune signaling in the pathogenesis of atopic dermatitis and consequences for treatments.Opposing Effects of Zac1 and Curcumin on AP-1-Regulated Expressions of S100A7 Improved epidermal barrier formation in human skin models by chitosan modulated dermal matrices.Monoclonal antibodies: the new magic bullets for allergy: IUPHAR Review 17.Interferon-γ Protects from Staphylococcal Alpha Toxin-Induced Keratinocyte Death through Apolipoprotein L1 Filaggrin inhibits generation of CD1a neolipid antigens by house dust mite-derived phospholipase.Topical Tetracycline Improves MC903-induced Atopic Dermatitis in Mice through Inhibition of Inflammatory Cytokines and Thymic Stromal Lymphopoietin Expression.Early pediatric atopic dermatitis shows only a cutaneous lymphocyte antigen (CLA)(+) TH2/TH1 cell imbalance, whereas adults acquire CLA(+) TH22/TC22 cell subsets Pathways to managing atopic dermatitis: consensus from the experts IL-23 induced in keratinocytes by endogenous TLR4 ligands polarizes dendritic cells to drive IL-22 responses to skin immunization Guanine nucleotide exchange factor RABGEF1 regulates keratinocyte-intrinsic signaling to maintain skin homeostasis IL-10 suppresses IL-17-mediated dermal inflammation and reduces the systemic burden of Vaccinia virus in a mouse model of eczema vaccinatum.Bee Venom Phospholipase A2 Ameliorates House Dust Mite Extract Induced Atopic Dermatitis Like Skin Lesions in Mice Adult Atopic Dermatitis with Comorbid Atopic Disease is Associated with Increased Risk of Infections: A Population-Based Cross-Sectional Study.

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Progressive activation of T(H)2/T(H)22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis.

http://www.wikidata.org/entity/Q37709681

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 August 2012

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Progressive activation of T

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Progressive activation of T(H) ...... and chronic atopic dermatitis.

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type

label

Progressive activation of T

@nl

Progressive activation of T(H) ...... and chronic atopic dermatitis.

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prefLabel

Progressive activation of T

@nl

Progressive activation of T(H) ...... and chronic atopic dermatitis.

@en

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J.JACI.2012.07.012

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The Journal of Allergy and Clinical Immunology

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Progressive activation of T(H) ...... and chronic atopic dermatitis.

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Avner Shemer

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Claire Q F Wang

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Cristina de Guzman Strong

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Emma Guttman-Yassky

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Hiroshi Mitsui

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Irma Cardinale

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James G Krueger

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Judilyn Fuentes-Duculan

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Julia K Gittler

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Kara J Gulewicz

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P2860

Expression patterns of S100A7 (psoriasin) and S100A9 (calgranulin-B) in keratinocyte differentiation

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling

Harshlight: a "corrective make-up" program for microarray chips.

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Phagocyte-specific S100 proteins: a novel group of proinflammatory molecules

Integrative responses to IL-17 and TNF-α in human keratinocytes account for key inflammatory pathogenic circuits in psoriasis

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

Genome-wide association study identifies three new susceptibility loci for adult asthma in the Japanese population

Adjusting batch effects in microarray expression data using empirical Bayes methods

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1344-1354

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scholarly article

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10.1016/J.JACI.2012.07.012

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6

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130

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2012-08-27T00:00:00Z

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230803755

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22951056

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3991245

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