Neutrophils select hypervirulent CovRS mutants of M1T1 group A Streptococcus during subcutaneous infection of mice
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Evolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Serotype M3 and M28 Group A Streptococci Have Distinct Capacities to Evade Neutrophil and TNF-α Responses and to Invade Soft Tissues.A Neutralizing Monoclonal IgG1 Antibody of Platelet-Activating Factor Acetylhydrolase SsE Protects Mice against Lethal Subcutaneous Group A Streptococcus InfectionType Six Secretion System of Bordetella bronchiseptica and Adaptive Immune Components Limit Intracellular Survival During InfectionContemporary Pharyngeal and Invasive emm1 and Invasive emm12 Group A Streptococcus Isolates Exhibit Similar In Vivo Selection for CovRS Mutants in Mice.The Mga Regulon but Not Deoxyribonuclease Sda1 of Invasive M1T1 Group A Streptococcus Contributes to In Vivo Selection of CovRS Mutations and Resistance to Innate Immune Killing Mechanisms.Multimerization of the Virulence-Enhancing Group A Streptococcus Transcription Factor RivR Is Required for Regulatory Activity.Streptolysin O Rapidly Impairs Neutrophil Oxidative Burst and Antibacterial Responses to Group A Streptococcus.Regulatory rewiring confers serotype-specific hyper-virulence in the human pathogen group A StreptococcusIdentifying protective Streptococcus pyogenes vaccine antigens recognized by both B and T cells in human adults and children.Null Mutations of Group A Streptococcus Orphan Kinase RocA: Selection in Mouse Infection and Comparison with CovS Mutations in Alteration of In Vitro and In Vivo Protease SpeB Expression and Virulence.RocA Is an Accessory Protein to the Virulence-Regulating CovRS Two-Component System in Group A Streptococcus.Regulatory gene mutation: a driving force behind group a Streptococcus strain- and serotype-specific variation.Combinatorial Synthetic Peptide Vaccine Strategy Protects against Hypervirulent CovR/S Mutant Streptococci.The Transcriptional Regulator CpsY Is Important for Innate Immune Evasion in Streptococcus pyogenes.The majority of 9,729 group A streptococcus strains causing disease secrete SpeB cysteine protease: pathogenesis implications.Requirement and Synergistic Contribution of Platelet-Activating Factor Acetylhydrolase Sse and Streptolysin S to Inhibition of Neutrophil Recruitment and Systemic Infection by Hypervirulent emm3 Group A Streptococcus in Subcutaneous Infection of MicHypervirulent Group A Streptococcus of Genotype emm3 Invades the Vascular System in Pulmonary Infection of Mice.Phenotypic Variation in the Group A Due to Natural Mutation of the Accessory Protein-Encoding GeneThe Roles of Two Type VI Secretion Systems in ATCC 12868Cytotoxicity and Survival Fitness of Invasive covS Mutant of Group A Streptococcus in Phagocytic Cells
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Neutrophils select hypervirulent CovRS mutants of M1T1 group A Streptococcus during subcutaneous infection of mice
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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name
Neutrophils select hypervirule ...... subcutaneous infection of mice
@en
Neutrophils select hypervirule ...... ubcutaneous infection of mice.
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type
label
Neutrophils select hypervirule ...... subcutaneous infection of mice
@en
Neutrophils select hypervirule ...... ubcutaneous infection of mice.
@nl
prefLabel
Neutrophils select hypervirule ...... subcutaneous infection of mice
@en
Neutrophils select hypervirule ...... ubcutaneous infection of mice.
@nl
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Neutrophils select hypervirule ...... subcutaneous infection of mice
@en
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Benfang Lei
Guanghui Liu
James A Wiley
Jinquan Li
Mengyao Liu
Wenchao Feng
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P304
P356
10.1128/IAI.01458-13
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P577
2014-01-22T00:00:00Z