Matrix metalloproteinase activity creates pro-angiogenic environment in primary human retinal pigment epithelial cells exposed to complement.
about
PEDF and its roles in physiological and pathological conditions: implication in diabetic and hypoxia-induced angiogenic diseasesSmoke exposure causes endoplasmic reticulum stress and lipid accumulation in retinal pigment epithelium through oxidative stress and complement activation.Why AMD is a disease of ageing and not of development: mechanisms and insights.Zinc supplementation inhibits complement activation in age-related macular degeneration.Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner.Local production of the alternative pathway component factor B is sufficient to promote laser-induced choroidal neovascularization.The role of complement in tumor growthAge-Related Macular Degeneration: A Disease of Systemic or Local Complement Dysregulation?A local complement response by RPE causes early-stage macular degeneration.Differential Expression of Complement Markers in Normal and AMD Transmitochondrial CybridsNitrite Modification of Extracellular Matrix Alters CD46 Expression and VEGF Release in Human Retinal Pigment Epithelium.A Targeted Inhibitor of the Alternative Complement Pathway Accelerates Recovery From Smoke-Induced Ocular InjuryMolecular response of chorioretinal endothelial cells to complement injury: implications for macular degeneration.C3a Increases VEGF and Decreases PEDF mRNA Levels in Human Retinal Pigment Epithelial CellsComplement Activation and Inhibition in Retinal Diseases.Neurodegeneration and Neuroinflammation in Diabetic Retinopathy: Potential Approaches to Delay Neuronal Loss.Activation of endogenously expressed ion channels by active complement in the retinal pigment epithelium.Pathway activation profiling reveals new insights into age-related macular degeneration and provides avenues for therapeutic interventions.Encapsulated Cell Technology-Based Delivery of a Complement Inhibitor Reduces Choroidal Neovascularization in a Mouse Model.Isolation, culture and characterization of primary mouse RPE cells.
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Matrix metalloproteinase activity creates pro-angiogenic environment in primary human retinal pigment epithelial cells exposed to complement.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 18 April 2012
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Matrix metalloproteinase activ ...... l cells exposed to complement.
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Matrix metalloproteinase activ ...... l cells exposed to complement.
@nl
type
label
Matrix metalloproteinase activ ...... l cells exposed to complement.
@en
Matrix metalloproteinase activ ...... l cells exposed to complement.
@nl
prefLabel
Matrix metalloproteinase activ ...... l cells exposed to complement.
@en
Matrix metalloproteinase activ ...... l cells exposed to complement.
@nl
P2860
P356
P1476
Matrix metalloproteinase activ ...... l cells exposed to complement.
@en
P2093
Bärbel Rohrer
Mausumi Bandyopadhyay
P2860
P304
P356
10.1167/IOVS.11-8638
P407
P577
2012-04-18T00:00:00Z