Leptin exacerbates sepsis-mediated morbidity and mortality.
about
Adipokines: biofactors from white adipose tissue. A complex hub among inflammation, metabolism, and immunity.Novel regulatory mechanisms for generation of the soluble leptin receptor: implications for leptin action.Adipocyte versus pituitary leptin in the regulation of pituitary hormones: somatotropes develop normally in the absence of circulating leptin.Alterations of leptin in the course of inflammation and severe sepsis.Cytokine responses to Staphylococcus aureus bloodstream infection differ between patient cohorts that have different clinical courses of infection.The impact of obesity on sepsis mortality: a retrospective review.High fat diet-induced obesity leads to proinflammatory response associated with higher expression of NOD2 protein.Endocrine dysfunction in sepsis: a beneficial or deleterious host response?Divergence of the response induced by xenogenic immunization in the sepsis survival of rats.Leptin and leptin receptor polymorphisms are associated with poor outcome (death) in patients with non-appendicular secondary peritonitis.Obesity enhances sepsis-induced liver inflammation and injury in miceAromadendrin Inhibits Lipopolysaccharide-Induced Nuclear Translocation of NF-κB and Phosphorylation of JNK in RAW 264.7 Macrophage Cells.Bone marrow leptin signaling mediates obesity-associated adipose tissue inflammation in male mice.Leptin as a link between the immune system and kidney-related diseases: leading actor or just a coadjuvant?Leptin fails to blunt the lipopolysaccharide-induced activation of the hypothalamic-pituitary-adrenal axis in rats.Leptin attenuates lipopolysaccharide or oleic acid-induced acute lung injury in mice.3,4,5-trihydroxycinnamic acid inhibits lipopolysaccharide (LPS)-induced inflammation by Nrf2 activation in vitro and improves survival of mice in LPS-induced endotoxemia model in vivo.Leptin independently predicts development of sepsis and its outcome.Leptin induces osteoblast differentiation of human valvular interstitial cells via the Akt and ERK pathways.
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Leptin exacerbates sepsis-mediated morbidity and mortality.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 02 June 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Leptin exacerbates sepsis-mediated morbidity and mortality.
@en
Leptin exacerbates sepsis-mediated morbidity and mortality.
@nl
type
label
Leptin exacerbates sepsis-mediated morbidity and mortality.
@en
Leptin exacerbates sepsis-mediated morbidity and mortality.
@nl
prefLabel
Leptin exacerbates sepsis-mediated morbidity and mortality.
@en
Leptin exacerbates sepsis-mediated morbidity and mortality.
@nl
P2093
P2860
P356
P1476
Leptin exacerbates sepsis-mediated morbidity and mortality.
@en
P2093
Eliyahu V Khankin
Kiichiro Yano
Matijs Van Meurs
Midori Yano
Nathan I Shapiro
Pedro R Castro
Samir M Parikh
Shou-Ching Shih
P2860
P304
P356
10.4049/JIMMUNOL.0903975
P407
P577
2010-06-02T00:00:00Z