Insulin-like growth factor 1 receptor-mediated cell survival in hypoxia depends on the promotion of autophagy via suppression of the PI3K/Akt/mTOR signaling pathway.
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L-3-n-Butylphthalide Activates Akt/mTOR Signaling, Inhibits Neuronal Apoptosis and Autophagy and Improves Cognitive Impairment in Mice with Repeated Cerebral Ischemia-Reperfusion Injury.Relation of the IGF/IGF1R system to autophagy in colitis and colorectal cancer.Bone marrow-derived mesenchymal stromal cells promote colorectal cancer cell death under low-dose irradiation.IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer
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Insulin-like growth factor 1 receptor-mediated cell survival in hypoxia depends on the promotion of autophagy via suppression of the PI3K/Akt/mTOR signaling pathway.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on March 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@en
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@nl
type
label
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@en
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@nl
prefLabel
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@en
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@nl
P2093
P2860
P921
P356
P1476
Insulin-like growth factor 1 r ...... 3K/Akt/mTOR signaling pathway.
@en
P2093
Jing-Zhi Guan
Ping Zhang
P2860
P304
P356
10.3892/MMR.2017.6265
P577
2017-03-01T00:00:00Z