Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer. - Wikidata - awgldk - TriplyDB

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

http://www.wikidata.org/entity/Q37726627

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Linking patient outcome to high throughput protein expression data identifies novel regulators of colorectal adenocarcinoma aggressiveness Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity.Epigenetic Regulation of Placenta-Specific 8 Contributes to Altered Function of Endothelial Colony-Forming Cells Exposed to Intrauterine Gestational Diabetes Mellitus.Claudin-Low Breast Cancer; Clinical & Pathological Characteristics Signaling pathway cooperation in TGF-β-induced epithelial-mesenchymal transition.NFAT1 Directly Regulates IL8 and MMP3 to Promote Melanoma Tumor Growth and Metastasis.Discovery of genes from feces correlated with colorectal cancer progression Three-dimensional culture system identifies a new mode of cetuximab resistance and disease-relevant genes in colorectal cancer.Castration Resistance in Prostate Cancer Is Mediated by the Kinase NEK6.Multiplexed imaging of intracellular protein networks.Overexpression of placenta specific 8 is associated with malignant progression and poor prognosis of clear cell renal cell carcinoma.TrkC promotes colorectal cancer growth and metastasis.An epithelial-to-mesenchymal transition-inducing potential of granulocyte macrophage colony-stimulating factor in colon cancer.Nerve growth factor and its receptors on onset and diagnosis of ovarian cancer Zinc finger protein X-linked is overexpressed in colorectal cancer and is associated with poor prognosis.ERK2 Mediates Metabolic Stress Response to Regulate Cell Fate Clustering of integrin α5 at the lateral membrane restores epithelial polarity in invasive colorectal cancer cells.lncRNA MIR100HG-derived miR-100 and miR-125b mediate cetuximab resistance via Wnt/β-catenin signaling.PLAC8, a new marker for human interstitial extravillous trophoblast cells, promotes their invasion and migration.Inhibition of autophagy prevents cadmium-induced prostate carcinogenesis.Knockout of the placenta specific 8 gene radiosensitizes nasopharyngeal carcinoma cells by activating the PI3K/AKT/GSK3β pathway.Identification of Differentially Expressed Genes between Original Breast Cancer and Xenograft Using Machine Learning Algorithms.Opportunities and Challenges in Implementation of Multiparameter Single Cell Analysis Platforms for Clinical Translation.The novel KLF4/PLAC8 signaling pathway regulates lung cancer growth.A Novel Role for the Interleukin-1 Receptor Axis in Resistance to Anti-EGFR Therapy

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Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

http://www.wikidata.org/entity/Q37726627

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on April 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@en

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@nl

type

label

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@en

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@nl

prefLabel

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@en

Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@nl

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Journal of Clinical Investigation

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Excess PLAC8 promotes an unconventional ERK2-dependent EMT in colon cancer.

@en

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Alina Starchenko

http://www.w3.org/2001/XMLSchema#string

Anne E Powell

http://www.w3.org/2001/XMLSchema#string

Cunxi Li

http://www.w3.org/2001/XMLSchema#string

Haiting Ma

http://www.w3.org/2001/XMLSchema#string

Keyur Desai

http://www.w3.org/2001/XMLSchema#string

Lila Solnica-Krezel

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Mary Kay Washington

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Michael J Gerdes

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Ramona Graves-Deal

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Robert J Coffey

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Extracellular regulated kinases (ERK) 1 and ERK2 are authentic substrates for the dual-specificity protein-tyrosine phosphatase VHR. A novel role in down-regulating the ERK pathway

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2172-2187

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scholarly article

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10.1172/JCI71103

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5

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124

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Gregory Daniel Ayers

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2014-04-01T00:00:00Z

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24691442

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4001536

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