Oxidative stress: a key contributor to diabetic cardiomyopathy. - Wikidata - awgldk - TriplyDB

Oxidative stress: a key contributor to diabetic cardiomyopathy.

Oxidative stress: a key contributor to diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q37731339

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Diabetic Cardiovascular Disease Induced by Oxidative Stress Resveratrol treatment reduces cardiac progenitor cell dysfunction and prevents morpho-functional ventricular remodeling in type-1 diabetic rats Loss of menin mediated by endothelial cells treated with CoPP is associated with increased maturation of adipocytes Mitochondrial Calpain-1 Disrupts ATP Synthase and Induces Superoxide Generation in Type 1 Diabetic Hearts: A Novel Mechanism Contributing to Diabetic Cardiomyopathy The effects of crocin, insulin and their co-administration on the heart function and pathology in streptozotocin-induced diabetic rats Heme oxygenase-1 prevents cardiac dysfunction in streptozotocin-diabetic mice by reducing inflammation, oxidative stress, apoptosis and enhancing autophagy High-glucose inhibits human fibroblast cell migration in wound healing via repression of bFGF-regulating JNK phosphorylation Conundrum of pathogenesis of diabetic cardiomyopathy: role of vascular endothelial dysfunction, reactive oxygen species, and mitochondria.Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin system.Liver X receptor agonist treatment attenuates cardiac dysfunction in type 2 diabetic db/db mice.Low molecular weight fucoidan alleviates cardiac dysfunction in diabetic Goto-Kakizaki rats by reducing oxidative stress and cardiomyocyte apoptosis.Increased systemic Th17 cytokines are associated with diastolic dysfunction in children and adolescents with diabetic ketoacidosis Oxidative Stress and Inflammation in Heart Disease: Do Antioxidants Have a Role in Treatment and/or Prevention?Intracellular calcium overloading and oxidative stress in cardiomyocyte necrosis via a mitochondriocentric signal-transducer-effector pathway.Neurohumoral stimulation.Hypothalamic paraventricular nucleus activation contributes to neurohumoral excitation in rats with heart failure Ginsenoside Rg1 ameliorates oxidative stress and myocardial apoptosis in streptozotocin-induced diabetic rats.Upregulation of arginase activity contributes to intracellular ROS production induced by high glucose in H9c2 cells Disturbances in calcium metabolism and cardiomyocyte necrosis: the role of calcitropic hormones.Oxidative stress contributes to the impaired sonic hedgehog pathway in type 1 diabetic mice with myocardial infarction.Adipose tissue biology and cardiomyopathy: translational implications.Activation of retinoid receptor-mediated signaling ameliorates diabetes-induced cardiac dysfunction in Zucker diabetic rats.Zinc Prevents the Development of Diabetic Cardiomyopathy in db/db Mice.Diabetic Cardiomyopathy: Mechanisms and Therapeutic Targets.Drosophila models of cardiac disease.Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.Direct renin inhibition prevents cardiac dysfunction in a diabetic mouse model: comparison with an angiotensin receptor antagonist and angiotensin-converting enzyme inhibitor Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes.Combination of Morroniside and Diosgenin Prevents High Glucose-Induced Cardiomyocytes Apoptosis.Matrine pretreatment improves cardiac function in rats with diabetic cardiomyopathy via suppressing ROS/TLR-4 signaling pathway.Cardiac ryanodine receptor in metabolic syndrome: is JTV519 (K201) future therapy?Taxifolin protects RPE cells against oxidative stress-induced apoptosis.Dihydromyricetin Protects against Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetic Mice.Mitochondrial quality-control dysregulation in conditional HO-1-/- mice.ROS removal by DJ-1: Arabidopsis as a new model to understand Parkinson's Disease.Metallothionein prevents diabetes-induced cardiac pathological changes, likely via the inhibition of succinyl-CoA:3-ketoacid coenzyme A transferase-1 nitration at Trp(374).Therapeutic effect of MG-132 on diabetic cardiomyopathy is associated with its suppression of proteasomal activities: roles of Nrf2 and NF-κB.Intrauterine hyperglycemia-induced inflammatory signalling via the receptor for advanced glycation end products in the cardiac muscle of the infants of diabetic mother rats.Aspalathin, a dihydrochalcone C-glucoside, protects H9c2 cardiomyocytes against high glucose induced shifts in substrate preference and apoptosis.Direct cardiovascular impact of SGLT2 inhibitors: mechanisms and effects.

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Oxidative stress: a key contributor to diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q37731339

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on March 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Oxidative stress: a key contributor to diabetic cardiomyopathy.

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Oxidative stress: a key contributor to diabetic cardiomyopathy.

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type

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Oxidative stress: a key contributor to diabetic cardiomyopathy.

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Oxidative stress: a key contributor to diabetic cardiomyopathy.

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prefLabel

Oxidative stress: a key contributor to diabetic cardiomyopathy.

@en

Oxidative stress: a key contributor to diabetic cardiomyopathy.

@nl

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Canadian Journal of Physiology and Pharmacology

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Oxidative stress: a key contributor to diabetic cardiomyopathy

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Abd Al-Rahman S Al-Shudiefat

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Ana Ludke

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Gursonika Binepal

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Pawan K Singal

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P2860

Activation of Nrf2 in defense against cadmium-induced oxidative stress

Insulin-dependent activation of endothelial nitric oxide synthase is impaired by O-linked glycosylation modification of signaling proteins in human coronary endothelial cells

Oxidative stress and the use of antioxidants in diabetes: linking basic science to clinical practice.

Global prevalence of diabetes: estimates for the year 2000 and projections for 2030

Meta-analysis: high-dosage vitamin E supplementation may increase all-cause mortality

Regulation and role of the mitochondrial transcription factor in the diabetic rat heart

Regulation of cardiomyocyte hypertrophy in diabetes at the transcriptional level

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Alterations in the diabetic myocardial proteome coupled with increased myocardial oxidative stress underlies diabetic cardiomyopathy

Effects of vitamins C and E and beta-carotene on the risk of type 2 diabetes in women at high risk of cardiovascular disease: a randomized controlled trial.

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233-240

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scholarly article

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10.1139/Y10-016

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3

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88

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2010-03-01T00:00:00Z

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20393588

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