Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis - Wikidata - awgldk - TriplyDB

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

http://www.wikidata.org/entity/Q37733555

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Dual inhibition of renin-angiotensin-aldosterone system and endothelin-1 in treatment of chronic kidney disease Endothelin Endothelin Blockade in Diabetic Kidney Disease Endothelin and the glomerulus in chronic kidney disease Improvements in the Management of Diabetic Nephropathy Crosstalk in glomerular injury and repair Natriuretic peptide receptor guanylyl cyclase-A pathway counteracts glomerular injury evoked by aldosterone through p38 mitogen-activated protein kinase inhibition.Amygdalar Endothelin-1 Regulates Pyramidal Neuron Excitability and Affects Anxiety Endothelial and platelet markers in diabetes mellitus type 2 Endothelial cell and podocyte autophagy synergistically protect from diabetes-induced glomerulosclerosis.Twenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy.Nuclear Factor Erythroid 2-Related Factor 2 Drives Podocyte-Specific Expression of Peroxisome Proliferator-Activated Receptor γ Essential for Resistance to Crescentic GN Influence of protein kinase C (PKC) on the prognosis of diabetic nephropathy patients.Diabetic nephropathy: landmark clinical trials and tribulations.One hundred ways to kill a podocyte.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.Endothelin-1 Induces Proteinuria by Heparanase-Mediated Disruption of the Glomerular Glycocalyx.Glomerular parietal epithelial cell activation induces collagen secretion and thickening of Bowman's capsule in diabetes.Endothelin antagonists in diabetic nephropathy: back to basics.Podocyte injury: the role of proteinuria, urinary plasminogen, and oxidative stress.Glomerular Endothelial Mitochondrial Dysfunction Is Essential and Characteristic of Diabetic Kidney Disease Susceptibility.Uric acid causes kidney injury through inducing fibroblast expansion, Endothelin-1 expression, and inflammation.Genetic and pharmacological inhibition of microRNA-92a maintains podocyte cell cycle quiescence and limits crescentic glomerulonephritis.Long-Term Endothelin-A Receptor Antagonism Provides Robust Renal Protection in Humanized Sickle Cell Disease Mice.Depletion of Gprc5a Promotes Development of Diabetic Nephropathy.Acute Kidney Injury and Progression of Diabetic Kidney Disease.COPD as an endothelial disorder: endothelial injury linking lesions in the lungs and other organs? (2017 Grover Conference Series).

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Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

http://www.wikidata.org/entity/Q37733555

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 April 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

@en

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis.

@nl

type

label

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

@en

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis.

@nl

prefLabel

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

@en

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis.

@nl

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P1433

Journal of the American Society of Nephrology

P1476

Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis

@en

P2093

Alain Schmitt

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David J Webb

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Jean-Marc Massé

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Marine Milon

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Masashi Yanagisawa

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Olivia Lenoir

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Sylvain Richard

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P2860

Collecting duct-specific knockout of the endothelin A receptor alters renal vasopressin responsiveness, but not sodium excretion or blood pressure

In response to protein load podocytes reorganize cytoskeleton and modulate endothelin-1 gene: implication for permselective dysfunction of chronic nephropathies

NFkappaB promotes inflammation, coagulation, and fibrosis in the aging glomerulus.

Mesangial cell, glomerular and renal vascular responses to endothelin in the rat kidney. Elucidation of signal transduction pathways

Addition of atrasentan to renin-angiotensin system blockade reduces albuminuria in diabetic nephropathy.

Wnt/β-catenin pathway in podocytes integrates cell adhesion, differentiation, and survival.

Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice.

Physiology and pathology of endothelin-1 in renal mesangium.

Epidermal growth factor receptor promotes glomerular injury and renal failure in rapidly progressive crescentic glomerulonephritis.

Mediators of diabetic renal disease: the case for tgf-Beta as the major mediator.

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1050-1062

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scholarly article

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10.1681/ASN.2013020195

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5

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25

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Pierre-Louis Tharaux

Yuri Kotelevtsev

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2014-04-10T00:00:00Z

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24722437

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4005294

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