Proinflammatory cytokines in heart failure: double-edged swords. - Wikidata - awgldk - TriplyDB

Proinflammatory cytokines in heart failure: double-edged swords.

Proinflammatory cytokines in heart failure: double-edged swords.

http://www.wikidata.org/entity/Q37734294

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Improving Cell Engraftment in Cardiac Stem Cell Therapy Hypothyroidism and its rapid correction alter cardiac remodeling Interaction between interleukin-1 beta and angiotensin II receptor 1 in hypothalamic paraventricular nucleus contributes to progression of heart failure Nutrition intervention to decrease symptoms in patients with advanced heart failure.IL-33 and ST2 levels in chronic kidney disease: Associations with inflammation, vascular abnormalities, cardiovascular events, and survival Interleukin-18 as a therapeutic target in acute myocardial infarction and heart failure.Pathogenesis and host response in Syrian hamsters following intranasal infection with Andes virus.Low-level laser therapy improves the inflammatory profile of rats with heart failure.mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy.Left Ventricular Dysfunction and CXCR3 Ligands in Hypertension: From Animal Experiments to a Population-Based Pilot Study Cardioprotective effect of ghrelin in cardiopulmonary bypass involves a reduction in inflammatory response Statin therapy blunts inflammatory activation and improves prognosis and left ventricular performance assessed by Tissue Doppler Imaging in subjects with chronic ischemic heart failure: results from the Daunia Heart Failure Registry.Depression, coronary artery disease, and physical activity: how much exercise is enough?Temporal and gefitinib-sensitive regulation of cardiac cytokine expression via chronic β-adrenergic receptor stimulation.Cardio-renal cachexia syndromes (CRCS): pathophysiological foundations of a vicious pathological circle Absence of SOCS3 in the cardiomyocyte increases mortality in a gp130-dependent manner accompanied by contractile dysfunction and ventricular arrhythmias.Inhibition of signal transducer and activator of transcription 3 (STAT3) attenuates interleukin-6 (IL-6)-induced collagen synthesis and resultant hypertrophy in rat heart.The Importance of NLRP3 Inflammasome in Heart Failure Changes in the monocytic subsets CD14(dim)CD16(+) and CD14(++)CD16(-) in chronic systolic heart failure patients.TLR3 deficiency induces chronic inflammatory cardiomyopathy in resistant mice following coxsackievirus B3 infection: role for IL-4.Could interferon-gamma be a therapeutic target for treating heart failure?IL-33 independently induces eosinophilic pericarditis and cardiac dilation: ST2 improves cardiac function Allostatic Load as a Complex Clinical Construct: A Case-Based Computational Modeling Approach Cardio-Ankle Vascular Index and C-Reactive Protein Are Useful Parameters for Identification of Ischemic Heart Disease in Acute Heart Failure Patients.The paradox of low BNP levels in obesity.Phosphoinositide-3 kinase signaling in cardiac hypertrophy and heart failure Inflammatory activation: cardiac, renal, and cardio-renal interactions in patients with the cardiorenal syndrome.Diagnosing destabilized heart failure in the emergency setting: current and future biomarker tests.Fibrosis and heart failure.Macrophages and islet inflammation in type 2 diabetes.Central nervous system circuits modified in heart failure: pathophysiology and therapeutic implications.Redox-sensitive mechanisms underlying vascular dysfunction in heart failure.Critical cytokine pathways to cardiac inflammation.IL-1β Inhibition in Cardiovascular Complications Associated to Diabetes Mellitus.Surgical optimization and characterization of a minimally invasive aortic banding procedure to induce cardiac hypertrophy in mice.Gene expression and levels of IL-6 and TNFα in PBMCs correlate with severity and functional class in patients with chronic heart failure.Interleukin-4 and Interleukin-10 Gene Polymorphisms in Patients with Inflammatory Bowel Disease.Fibronectin type III domain containing 5 expression in skeletal muscle in chronic heart failure-relevance of inflammatory cytokines.The Cardiomyocyte as a Source of Cytokines in Cardiac Injury.Appropriate candidates for statin use in heart failure.

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P2860

Proinflammatory cytokines in heart failure: double-edged swords.

http://www.wikidata.org/entity/Q37734294

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh

2010年學術文章

@zh-hant

name

Proinflammatory cytokines in heart failure: double-edged swords.

@en

Proinflammatory cytokines in heart failure: double-edged swords.

@nl

type

label

Proinflammatory cytokines in heart failure: double-edged swords.

@en

Proinflammatory cytokines in heart failure: double-edged swords.

@nl

prefLabel

Proinflammatory cytokines in heart failure: double-edged swords.

@en

Proinflammatory cytokines in heart failure: double-edged swords.

@nl

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Heart Failure Reviews

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Proinflammatory cytokines in heart failure: double-edged swords.

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Mohammad Jafar Mahmoudi

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Mona Hedayat

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Noel R Rose

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P2860

Interleukin-6-type cytokine signalling through the gp130/Jak/STAT pathway

Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy

NO-cGMP and TNF-alpha counter regulatory system in blood: understanding the mechanisms leading to myocardial dysfunction and failure.

Tumor necrosis factor-alpha upregulates angiotensin II type 1 receptors on cardiac fibroblasts.

The role of nitric oxide in cardiac depression induced by interleukin-1 beta and tumour necrosis factor-alpha.

Morphological and functional changes in cardiac myocytes isolated from mice overexpressing TNF-alpha.

Interleukin-1 beta inhibits phospholamban gene expression in cultured cardiomyocytes.

Glycoprotein 130 regulates cardiac myocyte survival in doxorubicin-induced apoptosis through phosphatidylinositol 3-kinase/Akt phosphorylation and Bcl-xL/caspase-3 interaction.

Nitric oxide-dependent and -independent mechanisms are involved in TNF-alpha -induced depression of cardiac myocyte contractility.

The anti-aggregating properties of vascular endothelium: interactions between prostacyclin and nitric oxide.

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