Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma. - Wikidata - awgldk - TriplyDB

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

http://www.wikidata.org/entity/Q37736273

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Brivanib, a dual FGF/VEGF inhibitor, is active both first and second line against mouse pancreatic neuroendocrine tumors developing adaptive/evasive resistance to VEGF inhibition EphB2 receptor controls proliferation/migration dichotomy of glioblastoma by interacting with focal adhesion kinase Antiangiogenic therapy of brain tumors: the role of bevacizumab.Vascular Mimicry: A Novel Neovascularization Mechanism Driving Anti-Angiogenic Therapy (AAT) Resistance in Glioblastoma Combined efficacy of cediranib and quinacrine in glioma is enhanced by hypoxia and causally linked to autophagic vacuole accumulation.Immunotherapy of brain cancers: the past, the present, and future directions.Potent and sustained inhibition of HIF-1α and downstream genes by a polyethyleneglycol-SN38 conjugate, EZN-2208, results in anti-angiogenic effects Surgical delivery of drug releasing poly(lactic-co-glycolic acid)/poly(ethylene glycol) paste with in vivo effects against glioblastoma.Synergistic Antivascular and Antitumor Efficacy with Combined Cediranib and SC6889 in Intracranial Mouse Glioma.CD105 (Endoglin) exerts prognostic effects via its role in the microvascular niche of paediatric high grade glioma.Bone marrow derived myeloid cells orchestrate antiangiogenic resistance in glioblastoma through coordinated molecular networks.Combination of vatalanib and a 20-HETE synthesis inhibitor results in decreased tumor growth in an animal model of human glioma.The timing of neural stem cell-based virotherapy is critical for optimal therapeutic efficacy when applied with radiation and chemotherapy for the treatment of glioblastoma Quinacrine synergistically enhances the antivascular and antitumor efficacy of cediranib in intracranial mouse glioma Activation of alternative pathways of angiogenesis and involvement of stem cells following anti-angiogenesis treatment in glioma.Therapeutic targeting of VEGF in the treatment of glioblastoma.Antiangiogenic therapies for glioblastoma.PVT1 affects growth of glioma microvascular endothelial cells by negatively regulating miR-186.Cancer Therapeutics Following Newton's Third Law The Different Effects of VEGFA121 and VEGFA165 on Regulating Angiogenesis Depend on Phosphorylation Sites of VEGFR2.Direct effect of bevacizumab on glioblastoma cell lines in vitro.Nuclear magnetic resonance imaging of tumour growth and neovasculature performance in vivo reveals Grb7 as a novel antiangiogenic target.Regulatory effects of Spirulina complex polysaccharides on growth of murine RSV-M glioma cells through Toll-like receptor 4.Survivin promotes glioma angiogenesis through vascular endothelial growth factor and basic fibroblast growth factor in vitro and in vivo.Vascular Mimicry: The Next Big Glioblastoma Target.VEGFR-2 Expression in Glioblastoma Multiforme Depends on Inflammatory Tumor Microenvironment.Hypoxic cell waves around necrotic cores in glioblastoma: a biomathematical model and its therapeutic implications.Toxicological effects of NCKU-21, a phenanthrene derivative, on cell growth and migration of A549 and CL1-5 human lung adenocarcinoma cells.Evaluation of Endoglin as an Angiogenesis Marker in Glioblastoma.Glioma cells enhance angiogenesis and inhibit endothelial cell apoptosis through the release of exosomes that contain long non-coding RNA CCAT2.Apparent diffusion coefficient histogram analysis stratifies progression-free survival in newly diagnosed bevacizumab-treated glioblastoma.Impact of Blood Vessel Quantity and Vascular Expression of CD133 and ICAM-1 on Survival of Glioblastoma Patients.Radiogenomic analysis of hypoxia pathway is predictive of overall survival in Glioblastoma.Major Challenges and Potential Microenvironment-Targeted Therapies in Glioblastoma.Hypoxia in Gliomas: Opening Therapeutical Opportunities Using a Mathematical-Based Approach.The Hedgehog signalling pathway and its prognostic impact in human gliomas.circ-SHKBP1 Regulates the Angiogenesis of U87 Glioma-Exposed Endothelial Cells through miR-544a/FOXP1 and miR-379/FOXP2 Pathways.

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Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

http://www.wikidata.org/entity/Q37736273

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 11 April 2010

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@en

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@nl

type

label

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@en

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@nl

prefLabel

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@en

Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

@nl

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Journal of oncology

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Antiangiogenic therapy and mechanisms of tumor resistance in malignant glioma.

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P2093

Cheryl Rahman

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Richard Grundy

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Ruman Rahman

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P2860

MiRNA-directed regulation of VEGF and other angiogenic factors under hypoxia

Vascular endothelial growth factor regulates endothelial cell survival through the phosphatidylinositol 3'-kinase/Akt signal transduction pathway. Requirement for Flk-1/KDR activation

Human tumstatin and human endostatin exhibit distinct antiangiogenic activities mediated by alpha v beta 3 and alpha 5 beta 1 integrins

VEGF-Trap: a VEGF blocker with potent antitumor effects.

Gene expression profiling reveals molecularly and clinically distinct subtypes of glioblastoma multiforme

Modes of resistance to anti-angiogenic therapy

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

Cell surface-localized matrix metalloproteinase-9 proteolytically activates TGF-beta and promotes tumor invasion and angiogenesis

The miR-17/92 polycistron is up-regulated in sonic hedgehog-driven medulloblastomas and induced by N-myc in sonic hedgehog-treated cerebellar neural precursors

miR-296 regulates growth factor receptor overexpression in angiogenic endothelial cells

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10.1155/2010/251231

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2010

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2010-04-11T00:00:00Z

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43299847

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