Complement driven innate immune response to malaria: fuelling severe malarial diseases.
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Compstatin Cp40 blocks hematin-mediated deposition of C3b fragments on erythrocytes: Implications for treatment of malarial anemiaTempol, an intracellular antioxidant, inhibits tissue factor expression, attenuates dendritic cell function, and is partially protective in a murine model of cerebral malaria.The complement systemPotential immune mechanisms associated with anemia in Plasmodium vivax malaria: a puzzling questionDetermining the population frequency of the CFHR3/CFHR1 deletion at 1q32.Cutting edge: the membrane attack complex of complement is required for the development of murine experimental cerebral malaria.Human antibodies fix complement to inhibit Plasmodium falciparum invasion of erythrocytes and are associated with protection against malariaComplement activation, placental malaria infection, and birth weight in areas characterized by unstable malaria transmission in central Sudan.Prospects and Pitfalls of Pregnancy-Associated Malaria Vaccination Based on the Natural Immune Response to Plasmodium falciparum VAR2CSA-Expressing Parasites.Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malariaExperimental Malaria in Pregnancy Induces Neurocognitive Injury in Uninfected Offspring via a C5a-C5a Receptor Dependent Pathway.Mutations of complement lectin pathway genes MBL2 and MASP2 associated with placental malariaThe C5 convertase is not required for activation of the terminal complement pathway in murine experimental cerebral malaria.Complement Activation in Placental Malaria.Dysregulation of the haem-haemopexin axis is associated with severe malaria in a case-control study of Ugandan children.Expression of complement and toll-like receptor pathway genes is associated with malaria severity in Mali: a pilot case control study.Functional roles for C5a and C5aR but not C5L2 in the pathogenesis of human and experimental cerebral malaria.Signatures of malaria-associated pathology revealed by high-resolution whole-blood transcriptomics in a rodent model of malaria.The immunological balance between host and parasite in malaria.Monocyte dysregulation and systemic inflammation during pediatric falciparum malaria.Mechanisms of naturally acquired immunity to P. falciparum and approaches to identify merozoite antigen targets.Effect of recombinant malarial antigen on monocyte functionality.A bite to fight: front-line innate immune defenses against malaria parasites.Plasmodium falciparum MSP3 exists in a complex on the merozoite surface and generates antibody response during natural infection.Complement Factor H Levels Associate With Malaria Susceptibility and Severity
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Complement driven innate immune response to malaria: fuelling severe malarial diseases.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 11 June 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@en
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
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type
label
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@en
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@nl
prefLabel
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@en
Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@nl
P2093
P2860
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Complement driven innate immune response to malaria: fuelling severe malarial diseases.
@en
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Chloe R McDonald
Karlee L Silver
Sarah J Higgins
P2860
P304
P356
10.1111/J.1462-5822.2010.01492.X
P50
P577
2010-06-11T00:00:00Z