A thalamo-hippocampal-ventral tegmental area loop may produce the positive feedback that underlies the psychotic break in schizophrenia.
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The reuniens and rhomboid nuclei: neuroanatomy, electrophysiological characteristics and behavioral implicationsCharacterizing thalamo-cortical disturbances in schizophrenia and bipolar illnessGluN2D N-Methyl-d-Aspartate Receptor Subunit Contribution to the Stimulation of Brain Activity and Gamma Oscillations by Ketamine: Implications for SchizophreniaAlterations in the hippocampus and thalamus in individuals at high risk for psychosis.On the Complexity of Brain Disorders: A Symptom-Based Approach.Disruption of mGluR5 in parvalbumin-positive interneurons induces core features of neurodevelopmental disorders.d-Cycloserine augmentation of cognitive remediation in schizophrenia.Abnormal stress responsivity in a rodent developmental disruption model of schizophreniaNicotine ameliorates NMDA receptor antagonist-induced deficits in contextual fear conditioning through high-affinity nicotinic acetylcholine receptors in the hippocampus.Progressive Decline in Hippocampal CA1 Volume in Individuals at Ultra-High-Risk for Psychosis Who Do Not Remit: Findings from the Longitudinal Youth at Risk Study.Converging on a core cognitive deficit: the impact of various neurodevelopmental insults on cognitive control.A Synaptic Function Approach to Investigating Complex Psychiatric Diseases.Increased hippocampal CA1 cerebral blood volume in schizophrenia.Coordinated activity of ventral tegmental neurons adapts to appetitive and aversive learning.Mediodorsal and visual thalamic connectivity differ in schizophrenia and bipolar disorder with and without psychosis history.Brain rhythms connect impaired inhibition to altered cognition in schizophrenia.Role of Thalamic Projection in NMDA Receptor-Induced Disruption of Cortical Slow Oscillation and Short-Term Plasticity.Transient inactivation of the neonatal ventral hippocampus permanently disrupts the mesolimbic regulation of prefrontal cholinergic transmission: implications for schizophreniaBridging Levels of Understanding in Schizophrenia Through Computational Modeling.Delta frequency optogenetic stimulation of the thalamic nucleus reuniens is sufficient to produce working memory deficits: relevance to schizophrenia.Fronto-limbic novelty processing in acute psychosis: disrupted relationship with memory performance and potential implications for delusions.Increased SNARE Protein-Protein Interactions in Orbitofrontal and Anterior Cingulate Cortices in SchizophreniaNeurophysiological differences between patients clinically at high risk for schizophrenia and neurotypical controls--first steps in development of a biomarker.NMDAR antagonist action in thalamus imposes δ oscillations on the hippocampusTransient inactivation of the neonatal ventral hippocampus impairs attentional set-shifting behavior: reversal with an α7 nicotinic agonist.Synaptic underpinnings of altered hippocampal function in glutaminase-deficient mice during maturation.Association of Thalamic Dysconnectivity and Conversion to Psychosis in Youth and Young Adults at Elevated Clinical Risk.Brain Biomarkers of Vulnerability and Progression to PsychosisThe Nucleus Reuniens of the Midline Thalamus Gates Prefrontal-Hippocampal Modulation of Ventral Tegmental Area Dopamine Neuron Activity.Connectivity, pharmacology, and computation: toward a mechanistic understanding of neural system dysfunction in schizophrenia.Targeting Neural Synchrony Deficits is Sufficient to Improve Cognition in a Schizophrenia-Related Neurodevelopmental Model.Excitation, inhibition, local oscillations, or large-scale loops: what causes the symptoms of schizophrenia?Neural oscillations during non-rapid eye movement sleep as biomarkers of circuit dysfunction in schizophrenia.Major diencephalic inputs to the hippocampus: supramammillary nucleus and nucleus reuniens. Circuitry and function.D-cycloserine in Schizophrenia: New Strategies for Improving Clinical Outcomes by Enhancing Plasticity.NMDA Receptor Internalization by Autoantibodies: A Reversible Mechanism Underlying Psychosis?Potential synergistic action of 19 schizophrenia risk genes in the thalamus.Multiscale modeling in the clinic: diseases of the brain and nervous system.Genetic deletion of fibroblast growth factor 14 recapitulates phenotypic alterations underlying cognitive impairment associated with schizophrenia.Dopamine in the thalamus: a hotbed for psychosis?
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A thalamo-hippocampal-ventral tegmental area loop may produce the positive feedback that underlies the psychotic break in schizophrenia.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 31 May 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@en
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@nl
type
label
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@en
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@nl
prefLabel
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@en
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@nl
P2093
P2860
P1476
A thalamo-hippocampal-ventral ...... chotic break in schizophrenia.
@en
P2093
Hyun Jae Pi
John E Lisman
Nonna A Otmakhova
Yuchun Zhang
P2860
P356
10.1016/J.BIOPSYCH.2010.04.007
P407
P577
2010-05-31T00:00:00Z