The RAGE axis in systemic inflammation, acute lung injury and myocardial dysfunction: an important therapeutic target?
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Small changes huge impact: the role of protein posttranslational modifications in cellular homeostasis and diseaseAnti-RAGE antibody ameliorates severe thermal injury in rats through regulating cellular immune function.The gene expression analysis of blood reveals S100A11 and AQP9 as potential biomarkers of infective endocarditis.Heterogeneous phenotypes of acute respiratory distress syndrome after major trauma.Year in review in Intensive Care Medicine 2010: I. Acute renal failure, outcome, risk assessment and ICU performance, sepsis, neuro intensive care and experimentalsBiomarkers in acute lung injury--marking forward progress.Retinol (Vitamin A) Increases α-Synuclein, β-Amyloid Peptide, Tau Phosphorylation and RAGE Content in Human SH-SY5Y Neuronal Cell Line.Plasma sRAGE enables prediction of acute lung injury after cardiac surgery in children.Identification of haplotype tag single nucleotide polymorphisms within the receptor for advanced glycation end products gene and their clinical relevance in patients with major trauma.Association between preoperative plasma sRAGE levels and recovery from cardiac surgery.High Mobility Group Box Protein 1 Boosts Endothelial Albumin Transcytosis through the RAGE/Src/Caveolin-1 Pathway.Participation of the receptor for advanced glycation end products in efferocytosis.Intensive care unit-acquired weakness: clinical phenotypes and molecular mechanisms.Role of myosin light chain and myosin light chain kinase in advanced glycation end product-induced endothelial hyperpermeability in vitro and in vivo.The impact of RAGE inhibition in animal models of bacterial sepsis: a systematic review and meta-analysis.RAGE inhibition reduces acute lung injury in mice.Therapeutic Effectiveness of Anti-RAGE Antibody Administration in a Rat Model of Crush Injury.Heat shock transcription factor 1 inhibits H₂O₂-induced cardiomyocyte death through suppression of high-mobility group box 1.Dietary advanced glycation end-products, its pulmonary receptor, and high mobility group box 1 in aspiration lung injury.A novel hypothesis: up-regulation of HO-1 by activation of PPARγ inhibits HMGB1-RAGE signaling pathway and ameliorates the development of ALI/ARDS.RAGE gene polymorphism in heart failure patients with and without angiographic evidence of significant coronary atherosclerosis.In silico assessment of S100A12 monomer and dimer structural dynamics: implications for the understanding of its metal-induced conformational changes.Receptor for advanced glycation end products axis in critically ill patients.Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.Small interfering RNA targeting receptor for advanced glycation end products protects the rats from multibacterial sepsis.
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The RAGE axis in systemic inflammation, acute lung injury and myocardial dysfunction: an important therapeutic target?
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 July 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The RAGE axis in systemic infl ...... important therapeutic target?
@en
The RAGE axis in systemic infl ...... important therapeutic target?
@nl
type
label
The RAGE axis in systemic infl ...... important therapeutic target?
@en
The RAGE axis in systemic infl ...... important therapeutic target?
@nl
prefLabel
The RAGE axis in systemic infl ...... important therapeutic target?
@en
The RAGE axis in systemic infl ...... important therapeutic target?
@nl
P2860
P1476
The RAGE axis in systemic infl ...... important therapeutic target?
@en
P2093
Anne Burke-Gaffney
Timothy W Evans
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P2888
P304
P356
10.1007/S00134-010-1952-Z
P577
2010-07-15T00:00:00Z