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The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?The "virgin birth", polyploidy, and the origin of cancerIncreased asymmetric and multi-daughter cell division in mechanically confined microenvironmentsIn vitro evidence for senescent multinucleated melanocytes as a source for tumor-initiating cells.Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal.Mos in the oocyte: how to use MAPK independently of growth factors and transcription to control meiotic divisions.Effect of X-Irradiation at Different Stages in the Cell Cycle on Individual Cell-Based Kinetics in an Asynchronous Cell Population.DNA damage-induced metaphase I arrest is mediated by the spindle assembly checkpoint and maternal age.Macroautophagy-aided elimination of chromatin: sorting of waste, sorting of fate?The roles of telomerase in the generation of polyploidy during neoplastic cell growth.Alternative lengthening of telomeres: recurrent cytogenetic aberrations and chromosome stability under extreme telomere dysfunction.Linking genomic reorganization to tumor initiation via the giant cell cycleThe development of a malignant tumor is due to a desperate asexual self-cloning process in which cancer stem cells develop the ability to mimic the genetic program of germline cells.KIF-2C expression is correlated with poor prognosis of operable esophageal squamous cell carcinoma male patients.Ionizing radiation-induced responses in human cells with differing TP53 status.Why it is crucial to analyze non clonal chromosome aberrations or NCCAs?Oncogenic cancer/testis antigens: prime candidates for immunotherapy.Do Multiwell Plate High Throughput Assays Measure Loss of Cell Viability Following Exposure to Genotoxic Agents?Multinucleated Giant Cancer Cells Produced in Response to Ionizing Radiation Retain Viability and Replicate Their Genome.Molecular and cytogenetic changes in multi-drug resistant cancer cells and their influence on new compounds testing.Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy Outcome.Genome reversion process of endopolyploidy confers chromosome instability on the descendent diploid cells.Respiration characteristics of mitochondria in parental and giant transformed cells of the murine Nemeth-Kellner lymphoma.Polyploid tumour cells elicit paradiploid progeny through depolyploidizing divisions and regulated autophagic degradation.Understanding aneuploidy in cancer through the lens of system inheritance, fuzzy inheritance and emergence of new genome systems.Staurosporine Induces the Generation of Polyploid Giant Cancer Cells in Non-Small-Cell Lung Carcinoma A549 Cells
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description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 02 August 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
MOS, aneuploidy and the ploidy cycle of cancer cells.
@en
MOS, aneuploidy and the ploidy cycle of cancer cells.
@nl
type
label
MOS, aneuploidy and the ploidy cycle of cancer cells.
@en
MOS, aneuploidy and the ploidy cycle of cancer cells.
@nl
prefLabel
MOS, aneuploidy and the ploidy cycle of cancer cells.
@en
MOS, aneuploidy and the ploidy cycle of cancer cells.
@nl
P2860
P356
P1433
P1476
MOS, aneuploidy and the ploidy cycle of cancer cells.
@en
P2093
J Erenpreisa
P2860
P2888
P304
P356
10.1038/ONC.2010.310
P407
P577
2010-08-02T00:00:00Z
P5875
P6179
1042424693