The blood-brain barrier, chemokines and multiple sclerosis
about
Early invasion of brain parenchyma by African trypanosomesMolecular biology, epidemiology, and pathogenesis of progressive multifocal leukoencephalopathy, the JC virus-induced demyelinating disease of the human brainPathophysiological impact of cigarette smoke exposure on the cerebrovascular system with a focus on the blood-brain barrier: expanding the awareness of smoking toxicity in an underappreciated areaPathobiology of tobacco smoking and neurovascular disorders: untied strings and alternative productsInnate immunity in the central nervous systemChemokine receptor CXCR2: physiology regulator and neuroinflammation controller?Neuroinflammation: a common pathway in CNS diseases as mediated at the blood-brain barrierMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesEndothelial Response to Glucocorticoids in Inflammatory DiseasesFeasibility of the use of combinatorial chemokine arrays to study blood and CSF in multiple sclerosisSystemic lipopolysaccharide compromises the blood-labyrinth barrier and increases entry of serum fluorescein into the perilymph.Evolution of the blood-brain barrier in newly forming multiple sclerosis lesionsMyeloid cell transmigration across the CNS vasculature triggers IL-1β-driven neuroinflammation during autoimmune encephalomyelitis in mice.A microfluidic device for measuring cell migration towards substrate-bound and soluble chemokine gradients.Tellurium compound AS101 ameliorates experimental autoimmune encephalomyelitis by VLA-4 inhibition and suppression of monocyte and T cell infiltration into the CNSChemokine receptor expression by inflammatory T cells in EAE.An in vitro blood-brain barrier model combining shear stress and endothelial cell/astrocyte co-culture.β-Arrestin1 and distinct CXCR4 structures are required for stromal derived factor-1 to downregulate CXCR4 cell-surface levels in neuroblastomaThe impact of microglial activation on blood-brain barrier in brain diseases.Imatinib ameliorates neuroinflammation in a rat model of multiple sclerosis by enhancing blood-brain barrier integrity and by modulating the peripheral immune responseBreaking down the barrier: the effects of HIV-1 on the blood-brain barrier.Macroglial plasticity and the origins of reactive astroglia in experimental autoimmune encephalomyelitis.In vitro blood-brain barrier models: current and perspective technologiesInflammatory cell trafficking across the blood-brain barrier: chemokine regulation and in vitro models.Does helminth activation of toll-like receptors modulate immune response in multiple sclerosis patients?Inflammatory response of endothelial cells to a human endogenous retrovirus associated with multiple sclerosis is mediated by TLR4.The nod-like receptor, Nlrp12, plays an anti-inflammatory role in experimental autoimmune encephalomyelitis.H(1)R expression by CD11B(+) cells is not required for susceptibility to experimental allergic encephalomyelitisAccelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis.Aurintricarboxylic acid ameliorates experimental autoimmune encephalomyelitis by blocking chemokine-mediated pathogenic cell migration and infiltration.Blood-brain barrier permeability of normal appearing white matter in relapsing-remitting multiple sclerosisTriggers and effectors of oxidative stress at blood-brain barrier level: relevance for brain ageing and neurodegenerationChemokine-mediated redirection of T cells constitutes a critical mechanism of glucocorticoid therapy in autoimmune CNS responses.The multifaceted responses of primary human astrocytes and brain microvascular endothelial cells to the Lyme disease spirochete, Borrelia burgdorferi.G protein-coupled receptors as therapeutic targets for multiple sclerosis.Design, Synthesis, and In Vitro and In Vivo Evaluation of an (18)F-Labeled Sphingosine 1-Phosphate Receptor 1 (S1P1) PET Tracer.Multiple sclerosis-induced neuropathic pain: pharmacological management and pathophysiological insights from rodent EAE modelsβ₂-adrenergic agonists modulate TNF-α induced astrocytic inflammatory gene expression and brain inflammatory cell populations.Peripheral VH4+ plasmablasts demonstrate autoreactive B cell expansion toward brain antigens in early multiple sclerosis patients.Alpha beta-crystallin expression and presentation following infection with murine gammaherpesvirus 68.
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The blood-brain barrier, chemokines and multiple sclerosis
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 August 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The blood-brain barrier, chemokines and multiple sclerosis
@en
The blood-brain barrier, chemokines and multiple sclerosis.
@nl
type
label
The blood-brain barrier, chemokines and multiple sclerosis
@en
The blood-brain barrier, chemokines and multiple sclerosis.
@nl
prefLabel
The blood-brain barrier, chemokines and multiple sclerosis
@en
The blood-brain barrier, chemokines and multiple sclerosis.
@nl
P2093
P2860
P1476
The blood-brain barrier, chemokines and multiple sclerosis
@en
P2093
David W Holman
Richard M Ransohoff
Robyn S Klein
P2860
P304
P356
10.1016/J.BBADIS.2010.07.019
P407
P577
2010-08-06T00:00:00Z