Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
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Imaging multiple sclerosis and other neurodegenerative diseasesTemporal and spatial increase of reactive nitrogen species in the kainate model of temporal lobe epilepsy.Mitochondria-ros crosstalk in the control of cell death and agingProtein redox modification as a cellular defense mechanism against tissue ischemic injury.Nitric oxide-mediated oxidative damage and the progressive demise of motor neurons in ALS.Sustained activation of Akt elicits mitochondrial dysfunction to block Plasmodium falciparum infection in the mosquito hostManganese superoxide dismutase: guardian of the powerhouse.Oxidative stress and microglial cells in Parkinson's diseaseCurrent Gaps in Understanding the Molecular Basis of FXTASOXPHOS mutations and neurodegenerationSerum Dihydrolipoamide Dehydrogenase Is a Labile Enzyme.Bioenergetic deficits in peripheral nerve sensory axons during chemotherapy-induced neuropathic pain resulting from peroxynitrite-mediated post-translational nitration of mitochondrial superoxide dismutase.Redox metabolism abnormalities in autistic children associated with mitochondrial diseaseRedox regulation of mitochondrial function with emphasis on cysteine oxidation reactions.Antioxidant and bioenergetic coupling between neurons and astrocytes.Cellular and molecular mechanisms of antioxidants in Parkinson's disease.Mitochondrial dysfunction in psychiatric and neurological diseases: cause(s), consequence(s), and implications of antioxidant therapy.Comparison of the Neuroprotective and Anti-Inflammatory Effects of the Anthocyanin Metabolites, Protocatechuic Acid and 4-Hydroxybenzoic Acid.Mitochondrial peroxiredoxin-5 as potential modulator of mitochondria-ER crosstalk in MPP+-induced cell death.Neuroprotective effects of umbelliferone and esculetin in a mouse model of Parkinson's disease.Post-Translational Oxidative Modifications of Mitochondrial Complex I (NADH: Ubiquinone Oxidoreductase): Implications for Pathogenesis and Therapeutics in Human Diseases.Rotenone-induced death of RGC-5 cells is caspase independent, involves the JNK and p38 pathways and is attenuated by specific green tea flavonoids.Role of SIRT1/PGC-1α in mitochondrial oxidative stress in autistic spectrum disorder.Established Principles and Emerging Concepts on the Interplay between Mitochondrial Physiology and S-(De)nitrosylation: Implications in Cancer and Neurodegeneration.Differential protection of black-seed oil on econucleotidase, cholinesterases and aminergic catabolizing enzyme in haloperidol-induced neuronal damage of male rats.Treatment with Hydrogen-Rich Saline Delays Disease Progression in a Mouse Model of Amyotrophic Lateral Sclerosis.Mitochondrial Complex I Reversible S-Nitrosation Improves Bioenergetics and Is Protective in Parkinson's Disease.
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P2860
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 September 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@en
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@nl
type
label
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@en
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@nl
prefLabel
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@en
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease.
@nl
P2860
P1476
Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease
@en
P2093
Shankar J Chinta
P2860
P356
10.3109/10715762.2010.509398
P577
2010-09-06T00:00:00Z