Viral infection and aging as cofactors for the development of pulmonary fibrosis.
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Gammaherpesviruses and pulmonary fibrosis: evidence from humans, horses, and rodentsPulmonary fibrosis: patterns and perpetratorsThe role of epidermal growth factor receptor (EGFR) signaling in SARS coronavirus-induced pulmonary fibrosis.Cellular senescence in normal and premature lung aging.Interstitial lung disease: NHLBI Workshop on the Primary Prevention of Chronic Lung Diseases.γ-Herpes virus-68, but not Pseudomonas aeruginosa or influenza A (H1N1), exacerbates established murine lung fibrosisInflammatory leukocyte phenotypes correlate with disease progression in idiopathic pulmonary fibrosis.Extensive phenotyping of individuals at risk for familial interstitial pneumonia reveals clues to the pathogenesis of interstitial lung disease.CD8+ T Cell Response to Gammaherpesvirus Infection Mediates Inflammation and Fibrosis in Interferon Gamma Receptor-Deficient MiceRight place, right time: the evolving role of herpesvirus infection as a "second hit" in idiopathic pulmonary fibrosisEmerging evidence for endoplasmic reticulum stress in the pathogenesis of idiopathic pulmonary fibrosis.Pulmonary fibrosis induced by γ-herpesvirus in aged mice is associated with increased fibroblast responsiveness to transforming growth factor-β.Six-SOMAmer Index Relating to Immune, Protease and Angiogenic Functions Predicts Progression in IPF.Lung carcinogenesis and fibrosis taken together: just coincidence?Negative regulation of lung inflammation and immunopathology by TNF-α during acute influenza infectionStreptococcus pneumoniae triggers progression of pulmonary fibrosis through pneumolysin.Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis.Flu and pulmonary fibrosis."Scar-cinoma": viewing the fibrotic lung mesenchymal cell in the context of cancer biology.Follow-up chest radiographic findings in patients with MERS-CoV after recovery.Human cystatin C: a new biomarker of idiopathic pulmonary fibrosis?Idiopathic pulmonary fibrosis: Epithelial-mesenchymal interactions and emerging therapeutic targets.Insufficient autophagy in idiopathic pulmonary fibrosis.The Toll-like receptor 3 L412F polymorphism and disease progression in idiopathic pulmonary fibrosis.
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P2860
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2010
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@en
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@nl
type
label
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@en
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@nl
prefLabel
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@en
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@nl
P2860
P356
P1476
Viral infection and aging as cofactors for the development of pulmonary fibrosis.
@en
P2093
Payal K Naik
P2860
P304
P356
10.1586/ERS.10.73
P577
2010-12-01T00:00:00Z