Focal adhesion kinase signaling and function in pancreatic cancer.
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Inhibiting the interaction of cMET and IGF-1R with FAK effectively reduces growth of pancreatic cancer cells in vitro and in vivoQuantitative proteomics with siRNA screening identifies novel mechanisms of trastuzumab resistance in HER2 amplified breast cancers.SRPX2 promotes cell migration and invasion via FAK dependent pathway in pancreatic cancer.The FAK scaffold inhibitor C4 disrupts FAK-VEGFR-3 signaling and inhibits pancreatic cancer growth.A small molecule FAK kinase inhibitor, GSK2256098, inhibits growth and survival of pancreatic ductal adenocarcinoma cells.Subgroup analysis reveals molecular heterogeneity and provides potential precise treatment for pancreatic cancers
P2860
Focal adhesion kinase signaling and function in pancreatic cancer.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on January 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Focal adhesion kinase signaling and function in pancreatic cancer.
@en
Focal adhesion kinase signaling and function in pancreatic cancer.
@nl
type
label
Focal adhesion kinase signaling and function in pancreatic cancer.
@en
Focal adhesion kinase signaling and function in pancreatic cancer.
@nl
prefLabel
Focal adhesion kinase signaling and function in pancreatic cancer.
@en
Focal adhesion kinase signaling and function in pancreatic cancer.
@nl
P2093
P356
P1476
Focal adhesion kinase signaling and function in pancreatic cancer.
@en
P2093
Deniz A Ucar
Long H Dang
Steven N Hochwald
P304
P356
10.2741/E283
P577
2011-01-01T00:00:00Z