about
NLRP6 in infection and inflammationIntegrating Inflammasome Signaling in Sexually Transmitted InfectionsTLR2 and RIP2 pathways mediate autophagy of Listeria monocytogenes via extracellular signal-regulated kinase (ERK) activationExosomal Hsp70 induces a pro-inflammatory response to foreign particles including mycobacteria.Reactive oxygen species regulate caspase-11 expression and activation of the non-canonical NLRP3 inflammasome during enteric pathogen infection.Role of the nlrp3 inflammasome in microbial infection.Toll or interleukin-1 receptor (TIR) domain-containing adaptor inducing interferon-β (TRIF)-mediated caspase-11 protease production integrates Toll-like receptor 4 (TLR4) protein- and Nlrp3 inflammasome-mediated host defense against enteropathogensReceptor interacting protein kinase 2-mediated mitophagy regulates inflammasome activation during virus infectionThe NOD-like receptor NLRP12 attenuates colon inflammation and tumorigenesis.FADD and caspase-8 mediate priming and activation of the canonical and noncanonical Nlrp3 inflammasomes.Exosomal membrane molecules are potent immune response modulators.Immunity to uropathogens: the emerging roles of inflammasomes.Regulation of PPAR-gamma gene in human promyelocytic HL-60 cell line.Autophagy modulates Borrelia burgdorferi-induced production of interleukin-1β (IL-1β)Vitamin D3-dependent pathway regulates TACO gene transcription.Cholesterol-sensor initiates M. tuberculosis entry into human macrophages.Lipids regulate P2X7-receptor-dependent actin assembly by phagosomes via ADP translocation and ATP synthesis in the phagosome lumen.Sphingosine-1-phosphate receptors stimulate macrophage plasma-membrane actin assembly via ADP release, ATP synthesis and P2X7R activation.Initial receptor-ligand interactions modulate gene expression and phagosomal properties during both early and late stages of phagocytosis.Green tea polyphenol inhibits Mycobacterium tuberculosis survival within human macrophages.Targeting NLRP6 to enhance immunity against bacterial infections.Trafficking of cholesterol to the ER is required for NLRP3 inflammasome activationRight place, right time: localisation and assembly of the NLRP3 inflammasomeFunctional genomics of PPAR-gamma in human immunomodulatory cellsCommon Differences: The Ability of Inflammasomes to Distinguish Between Self and Pathogen Nucleic Acids During InfectionAdapt(ed) to repair - TH2 immune responses in the bladder promote recurrent infectionsLipids, inflammasomes, metabolism, and disease
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Paras K Anand
@nl
Paras K Anand
@sl
Paras K. Anand
@en
Paras K. Anand
@es
type
label
Paras K Anand
@nl
Paras K Anand
@sl
Paras K. Anand
@en
Paras K. Anand
@es
prefLabel
Paras K Anand
@nl
Paras K Anand
@sl
Paras K. Anand
@en
Paras K. Anand
@es
P106
P1153
7102973453
P31
P496
0000-0003-2503-3653