about
Quantitative analysis of exercise-induced enhancement of early- and late-systolic retrograde coronary blood flow.Sex differences in pulmonary vascular control: focus on the nitric oxide pathwayGuiding the failing heart to exercise.Exercise limits the production of endothelin in the coronary vasculatureCoronary-aortic interaction during ventricular isovolumic contraction.Left ventricular remodeling in swine after myocardial infarction: a transcriptional genomics approach.Vagal nerve stimulation started just prior to reperfusion limits infarct size and no-reflowExercise hyperaemia in the heart: the search for the dilator mechanism.Sarcomeric dysfunction in heart failure.Myofilament dysfunction in cardiac disease from mice to men.Familial hypercholesterolemia impairs exercise-induced systemic vasodilation due to reduced NO bioavailability.Phosphodiesterase-5 activity exerts a coronary vasoconstrictor influence in awake swine that is mediated in part via an increase in endothelin production.Pulmonary vasoconstrictor influence of endothelin in exercising swine depends critically on phosphodiesterase 5 activity.Sympathetic coronary vasomotor control: are women really the weaker sex?'Integrative Physiology 2.0': integration of systems biology into physiology and its application to cardiovascular homeostasis.Regulation of coronary resistance vessel tone in response to exercise.Peripheral circulation.Stress myocardial perfusion: imaging with multidetector CT.Exercise training in adverse cardiac remodeling.Regulation of coronary blood flow in health and ischemic heart disease.Gene reprogramming in exercise-induced cardiac hypertrophy in swine: A transcriptional genomics approach.Altered purinergic signaling in uridine adenosine tetraphosphate-induced coronary relaxation in swine with metabolic derangement.Mitochondrial adaptations within chronically ischemic swine myocardium.Pulmonary arteriovenous malformations: haemodynamics and shunt closure.Hybrid contact force ablation: best of both worlds?Integrated control of pulmonary vascular tone by endothelin and angiotensin II in exercising swine depends on gender.Alterations in vasomotor control of coronary resistance vessels in remodelled myocardium of swine with a recent myocardial infarction.Both beta1- and beta2-adrenoceptors contribute to feedforward coronary resistance vessel dilation during exercise.Prevention of myofilament dysfunction by beta-blocker therapy in postinfarct remodeling.Uridine adenosine tetraphosphate is a novel vasodilator in the coronary microcirculation which acts through purinergic P1 but not P2 receptors.Contribution of endothelin and its receptors to the regulation of vascular tone during exercise is different in the systemic, coronary and pulmonary circulation.Quantification of myocardial blood flow by adenosine-stress CT perfusion imaging in pigs during various degrees of stenosis correlates well with coronary artery blood flow and fractional flow reserve.In vivo role of heme oxygenase in ischemic coronary vasodilation.Contribution of endothelin to coronary vasomotor tone is abolished after myocardial infarction.Contribution of KATP+ channels to coronary vasomotor tone regulation is enhanced in exercising swine with a recent myocardial infarction.Cardiac myocytes control release of endothelin-1 in coronary vasculature.Exercise unmasks autonomic dysfunction in swine with a recent myocardial infarction.Phosphodiesterase 5 inhibition-induced coronary vasodilation is reduced after myocardial infarction.Nitroso-redox balance in control of coronary vasomotor tone.Pulmonary blood flow and its distribution in highly trained endurance athletes and healthy control subjects.
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description
hulumtuese
@sq
researcher
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wetenschapper
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հետազոտող
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name
Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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type
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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prefLabel
Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
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Daphne Merkus
@sl
P106
P108
P1153
6603382866
P21
P31
P496
0000-0002-4852-831X