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Safety and T cell modulating effects of high dose vitamin D3 supplementation in multiple sclerosisThe multiple sclerosis susceptibility genes TAGAP and IL2RA are regulated by vitamin D in CD4+ T cells.Vitamin D status is positively correlated with regulatory T cell function in patients with multiple sclerosis.Vitamin D Status Does Not Affect Disability Progression of Patients with Multiple Sclerosis over Three Year Follow-Up.Vitamin D as an immune modulator in multiple sclerosis, a review.The relevance of vitamin D receptor gene polymorphisms for vitamin D research in multiple sclerosis.Complementary and alternative medicine in alopecia areata.Isolation of primary microglia from the human post-mortem brain: effects of ante- and post-mortem variables.Vitamin d and multiple sclerosis: correlation, causality, and controversyVitamin D as a T-cell modulator in multiple sclerosis.Effects of vitamin D on the peripheral adaptive immune system: a review.Vitamin D in the healthy and inflamed central nervous system: access and function.Vitamin D in multiple sclerosis: implications for assessment and treatment.Efficacy of vitamin D3 as add-on therapy in patients with relapsing-remitting multiple sclerosis receiving subcutaneous interferon β-1a: a Phase II, multicenter, double-blind, randomized, placebo-controlled trial.A low vitamin D status at diagnosis is associated with an early conversion to secondary progressive multiple sclerosis.Network of nuclear receptor ligands in multiple sclerosis: Common pathways and interactions of sex-steroids, corticosteroids and vitamin D3-derived molecules.Characteristics of differentiated CD8(+) and CD4 (+) T cells present in the human brain.Intracellular IL-10 detection in T cells by flowcytometry: the use of protein transport inhibitors revisited.Effect of vitamin D(3) supplementation on peripheral B cell differentiation and isotype switching in patients with multiple sclerosis.Exploring the effect of vitamin D3 supplementation on the anti-EBV antibody response in relapsing-remitting multiple sclerosis.GM-CSF production by CD4+ T cells in MS patients: regulation by regulatory T cells and vitamin D.Increased inflammasome related gene expression profile in PBMC may facilitate T helper 17 cell induction in multiple sclerosis.Association study on two vitamin D receptor gene polymorphisms and vitamin D metabolites in multiple sclerosis.Vitamin D supplementation and antibodies against the Epstein-Barr virus in multiple sclerosis patients.Microglia in normal appearing white matter of multiple sclerosis are alerted but immunosuppressed.Association of vitamin D metabolite levels with relapse rate and disability in multiple sclerosis.Vitamin D3 supplementation and the IL-2/IL-2R pathway in multiple sclerosis: Attenuation of progressive disturbances?On the ethics of not supplementing low 25-hydroxyvitamin D levels in a controlled study in relapsing remitting multiple sclerosis.Vitamin D3 supplementation in multiple sclerosis: Symptoms and biomarkers of depression.Circulating vitamin D binding protein levels are not associated with relapses or with vitamin D status in multiple sclerosis.Immune regulatory effects of high dose vitamin D3 supplementation in a randomized controlled trial in relapsing remitting multiple sclerosis patients receiving IFNβ; the SOLARIUM study.Vitamin D status is negatively correlated with retinal nerve fiber layer thickness in relapsing-remitting MS patients without acute optic neuritis.Expression of vitamin D receptor and metabolizing enzymes in multiple sclerosis-affected brain tissue.Phenotyping primary human microglia: tight regulation of LPS responsiveness.Vitamin D status in patients with MS is negatively correlated with depression, but not with fatigue.Immunomodulation by vitamin D in multiple sclerosis: More than IL-17.Fraction of IL-10+ and IL-17+ CD8 T cells is increased in MS patients in remission and during a relapse, but is not influenced by immune modulators.Fatigue and heat sensitivity in patients with multiple sclerosis.Th17 expansion in MS patients is counterbalanced by an expanded CD39+ regulatory T cell population during remission but not during relapse.Reduction in IL-10 producing B cells (Breg) in multiple sclerosis is accompanied by a reduced naïve/memory Breg ratio during a relapse but not in remission.
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description
hulumtues
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onderzoeker
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հետազոտող
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name
Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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type
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Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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prefLabel
Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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Joost Smolders
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P214
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P1053
B-7658-2009
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P1153
23101501900
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0000 0003 9593 7498
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P3829
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