Targeting mitochondrial function for the treatment of acute spinal cord injury. - Wikidata - awgldk - TriplyDB

Targeting mitochondrial function for the treatment of acute spinal cord injury.

Targeting mitochondrial function for the treatment of acute spinal cord injury.

http://www.wikidata.org/entity/Q37848238

about

Skeletal muscle mitochondrial health and spinal cord injury Imaging mass spectrometry reveals loss of polyunsaturated cardiolipins in the cortical contusion, hippocampus, and thalamus after traumatic brain injury Ginsenoside Rd attenuates mitochondrial permeability transition and cytochrome C release in isolated spinal cord mitochondria: involvement of kinase-mediated pathways.Neuroprotective effects of N-acetyl-cysteine and acetyl-L-carnitine after spinal cord injury in adult rats.Changes to mitochondrial ultrastructure in optic nerve vulnerable to secondary degeneration in vivo are limited by irradiation at 670 nm Batroxobin protects against spinal cord injury in rats by promoting the expression of vascular endothelial growth factor to reduce apoptosis.Neuroprotective effects of exendin-4 in rat model of spinal cord injury via inhibiting mitochondrial apoptotic pathway.Acetyl-L-carnitine treatment following spinal cord injury improves mitochondrial function correlated with remarkable tissue sparing and functional recovery.Intrathecal Acetyl-L-Carnitine Protects Tissue and Improves Function after a Mild Contusive Spinal Cord Injury in Rats.Vitamins and nutrients as primary treatments in experimental brain injury: Clinical implications for nutraceutical therapies.The effect of cigarette smoke exposure on spinal cord injury in rats Role of mitochondria in regulating microRNA activity and its relevance to the central nervous system Provision of nutrients after acute spinal cord injury: the implications of feast and famine.Antioxidant strategies in the management of diabetic neuropathy.Targeting the NLRP3 inflammasome to attenuate spinal cord injury in mice.Neurotherapeutics. Editorial.Pioglitazone treatment following spinal cord injury maintains acute mitochondrial integrity and increases chronic tissue sparing and functional recovery.Mitochondrial-Based Therapeutics for the Treatment of Spinal Cord Injury: Mitochondrial Biogenesis as a Potential Pharmacological Target.Therapeutic efficacy of cyclosporin A against spinal cord injury in rats with hyperglycemia.Post-Injury Treatment with NIM811 Promotes Recovery of Function in Adult Female Rats after Spinal Cord Contusion: A Dose-Response Study.Identification of Dual Mechanisms Mediating 5-hydroxytryptamine Receptor 1F Induced Mitochondrial Biogenesis.Differential proteomic analysis of acute contusive spinal cord injury in rats using iTRAQ reagent labeling and LC-MS/MS.Synthesis of methylprednisolone loaded ibuprofen modified dextran based nanoparticles and their application for drug delivery in acute spinal cord injury.D-β-hydroxybutyrate promotes functional recovery and relieves pain hypersensitivity in mice with spinal cord injury.Mitochondrial Division Inhibitor 1 Ameliorates Mitochondrial Injury, Apoptosis, and Motor Dysfunction After Acute Spinal Cord Injury in Rats.Targeting the mitochondrial permeability transition pore in traumatic central nervous system injury

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Targeting mitochondrial function for the treatment of acute spinal cord injury.

http://www.wikidata.org/entity/Q37848238

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on April 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@en

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@nl

type

label

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@en

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@nl

prefLabel

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@en

Targeting mitochondrial function for the treatment of acute spinal cord injury.

@nl

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S13311-011-0031-7

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Neurotherapeutics

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Targeting mitochondrial function for the treatment of acute spinal cord injury.

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Alexander G Rabchevsky

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Joe E Springer

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Melanie L McEwen

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Patrick G Sullivan

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P2860

Debio 025, a cyclophilin binding molecule, is highly efficient in clearing hepatitis C virus (HCV) replicon-containing cells when used alone or in combination with specifically targeted antiviral therapy for HCV (STAT-C) inhibitors

NIM811, a Cyclophilin Inhibitor, Exhibits Potent In Vitro Activity against Hepatitis C Virus Alone or in Combination with Alpha Interferon

Combinations of Cyclophilin Inhibitor NIM811 with Hepatitis C Virus NS3-4A Protease or NS5B Polymerase Inhibitors Enhance Antiviral Activity and Suppress the Emergence of Resistance

The Isomerase Active Site of Cyclophilin A Is Critical for Hepatitis C Virus Replication

Structural and biochemical basis of apoptotic activation by Smac/DIABLO

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

Mitochondrial participation in ischemic and traumatic neural cell death

Endonuclease G is an apoptotic DNase when released from mitochondria

Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane

Apoptosis-inducing factor (AIF): a novel caspase-independent death effector released from mitochondria

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s13311-011-0031-7

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168-179

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scholarly article

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10.1007/S13311-011-0031-7

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2

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21360236

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