Neuronal hyperexcitability: a substrate for central neuropathic pain after spinal cord injury.
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Pathophysiology, Clinical Importance, and Management of Neurogenic Lower Urinary Tract Dysfunction Caused by Suprasacral Spinal Cord InjuryOverexpression of the astrocyte glutamate transporter GLT1 exacerbates phrenic motor neuron degeneration, diaphragm compromise, and forelimb motor dysfunction following cervical contusion spinal cord injury.Chronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.Persistent at-level thermal hyperalgesia and tactile allodynia accompany chronic neuronal and astrocyte activation in superficial dorsal horn following mouse cervical contusion spinal cord injuryGap junction proteins and their role in spinal cord injuryDysregulation of Kv3.4 channels in dorsal root ganglia following spinal cord injuryActivation of spinal and supraspinal cannabinoid-1 receptors leads to antinociception in a rat model of neuropathic spinal cord injury painCombinations of intrathecal gamma-amino-butyrate receptor agonists and N-methyl-d-aspartate receptor antagonists in rats with neuropathic spinal cord injury pain.Neuropathic pain prevalence following spinal cord injury: A systematic review and meta-analysis.Decreased spinothalamic and dorsal column medial lemniscus-mediated function is associated with neuropathic pain after spinal cord injury.GLT1 overexpression reverses established neuropathic pain-related behavior and attenuates chronic dorsal horn neuron activation following cervical spinal cord injury.Astrocytic CX43 hemichannels and gap junctions play a crucial role in development of chronic neuropathic pain following spinal cord injury.Recombinant neural progenitor transplants in the spinal dorsal horn alleviate chronic central neuropathic pain.Spinal cord injury pain: mechanisms and management.Opioid administration following spinal cord injury: implications for pain and locomotor recovery.Circulating miR-23b-3p, miR-145-5p and miR-200b-3p are potential biomarkers to monitor acute pain associated with laminitis in horses.Intrathecal Transplantation of Embryonic Stem Cell-Derived Spinal GABAergic Neural Precursor Cells Attenuates Neuropathic Pain in a Spinal Cord Injury Rat Model.Opposite, bidirectional shifts in excitation and inhibition in specific types of dorsal horn interneurons are associated with spasticity and pain post-SCI.Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.Enhancing excitatory activity of somatosensory cortex alleviates neuropathic pain through regulating homeostatic plasticity.Combination Drug Therapy for Pain following Chronic Spinal Cord Injury.A decerebrate adult mouse model for examining the sensorimotor control of locomotion.Ablation of IB4 non-peptidergic afferents in the rat facet joint prevents injury-induced pain and thalamic hyperexcitability via supraspinal glutamate transporters.Intra-spinal microstimulation may alleviate chronic pain after spinal cord injury.How is chronic pain related to sympathetic dysfunction and autonomic dysreflexia following spinal cord injury?Multidimensional clinical pain phenotypes after spinal cord injury.
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Neuronal hyperexcitability: a substrate for central neuropathic pain after spinal cord injury.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on June 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
@en
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
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type
label
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
@en
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
@nl
prefLabel
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
@en
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
@nl
P2860
P1476
Neuronal hyperexcitability: a ...... pain after spinal cord injury.
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P2093
Claire E Hulsebosch
Young Seob Gwak
P2860
P2888
P304
P356
10.1007/S11916-011-0186-2
P407
P577
2011-06-01T00:00:00Z