Multiple sclerosis as a neurodegenerative disease: pathology, mechanisms and therapeutic implications.
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Imaging multiple sclerosis and other neurodegenerative diseasesRole of microglia in CNS inflammationMetformin treatment has no beneficial effect in a dose-response survival study in the SOD1(G93A) mouse model of ALS and is harmful in female miceA combined post-mortem magnetic resonance imaging and quantitative histological study of multiple sclerosis pathology.Sex-dependent treatment of chronic EAE with partial MHC class II constructs.The Effects of Blast Exposure on Protein Deimination in the BrainA switch in pathogenic mechanism in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in IFN-γ-inducible lysosomal thiol reductase-free mice.Identification of cellular infiltrates during early stages of brain inflammation with magnetic resonance microscopy.Search for cellular stress biomarkers in lymphocytes from patients with multiple sclerosis: a pilot studyInsights into the evolutionary features of human neurodegenerative diseases.Hereditary diffuse leukoencephalopathy with spheroids with phenotype of primary progressive multiple sclerosisDerivation of neural stem cells from human adult peripheral CD34+ cells for an autologous model of neuroinflammation.Tissue transglutaminase in marmoset experimental multiple sclerosis: discrepancy between white and grey matter.Hereditary diffuse leukoencephalopathy with axonal spheroids (HDLS): a misdiagnosed disease entity.Total-tau in cerebrospinal fluid of patients with multiple sclerosis decreases in secondary progressive stage of disease and reflects degree of brain atrophy.The Potential of Metabolic Imaging.Cerebrospinal fluid lactate is associated with multiple sclerosis disease progression.Visualizing brain inflammation with a shingled-leg radio-frequency head probe for 19F/1H MRI.Clonally expanded mitochondrial DNA deletions within the choroid plexus in multiple sclerosis.Cytokine and chemokine alterations in tissue, CSF, and plasma in early presymptomatic phase of experimental allergic encephalomyelitis (EAE), in a rat model of multiple sclerosis.Assessing relapses and response to relapse treatment in patients with multiple sclerosis: a nursing perspective.Antecedents of Coping with the Disease in Patients with Multiple Sclerosis: A Qualitative Content Analysis.Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.Elevated axonal membrane permeability and its correlation with motor deficits in an animal model of multiple sclerosisMesenchymal Stem Cells of Dental Origin-Their Potential for Antiinflammatory and Regenerative Actions in Brain and Gut Damage.Laquinimod, a once-daily oral drug in development for the treatment of relapsing-remitting multiple sclerosis.Mitochondrial sirtuins--a new therapeutic target for repair and protection in multiple sclerosis.The role of mitochondria in axonal degeneration and tissue repair in MS.Initial immunopathogenesis of multiple sclerosis: innate immune response.Relapsing and progressive forms of multiple sclerosis: insights from pathology.Mitochondrial dysfunction in central nervous system white matter disorders.Cognitive impairment in neuro-Behcet's disease and multiple sclerosis: a comparative study.Solid lipid nanoparticles surface modified with anti-Contactin-2 or anti-Neurofascin for brain-targeted delivery of medicines.Myelin Basic Protein Citrullination in Multiple Sclerosis: A Potential Therapeutic Target for the Pathology.Vitamin D therapy in experimental allergic encephalomyelitis could be limited by opposing effects of sphingosine 1-phosphate and gelsolin dysregulation.Nogo-A Antibodies for Progressive Multiple Sclerosis.The sphingosine-1-phosphate receptor: A novel therapeutic target for multiple sclerosis and other autoimmune diseases.Short-term cuprizone feeding verifies N-acetylaspartate quantification as a marker of neurodegeneration.Lipidome analysis in multiple sclerosis reveals protein lipoxidative damage as a potential pathogenic mechanism.The Role of Galectin-3: From Oligodendroglial Differentiation and Myelination to Demyelination and Remyelination Processes in a Cuprizone-Induced Demyelination Model.
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Multiple sclerosis as a neurodegenerative disease: pathology, mechanisms and therapeutic implications.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Multiple sclerosis as a neurod ...... and therapeutic implications.
@en
Multiple sclerosis as a neurod ...... and therapeutic implications.
@nl
type
label
Multiple sclerosis as a neurod ...... and therapeutic implications.
@en
Multiple sclerosis as a neurod ...... and therapeutic implications.
@nl
prefLabel
Multiple sclerosis as a neurod ...... and therapeutic implications.
@en
Multiple sclerosis as a neurod ...... and therapeutic implications.
@nl
P921
P1476
Multiple sclerosis as a neurod ...... s and therapeutic implications
@en
P2093
Christine Stadelmann
P304
P356
10.1097/WCO.0B013E328346056F
P577
2011-06-01T00:00:00Z