Chronic perivascular inoculation with Chlamydophila pneumoniae results in plaque formation in vivo. - Wikidata - awgldk - TriplyDB

Chronic perivascular inoculation with Chlamydophila pneumoniae results in plaque formation in vivo.

Chronic perivascular inoculation with Chlamydophila pneumoniae results in plaque formation in vivo.

http://www.wikidata.org/entity/Q37862676

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Vascular cells contribute to atherosclerosis by cytokine- and innate-immunity-related inflammatory mechanisms.Pro-atherogenic lung and oral pathogens induce an inflammatory response in human and mouse mast cells

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Chronic perivascular inoculation with Chlamydophila pneumoniae results in plaque formation in vivo.

http://www.wikidata.org/entity/Q37862676

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2006

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chronic perivascular inoculati ...... s in plaque formation in vivo.

@en

Chronic perivascular inoculati ...... s in plaque formation in vivo.

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type

label

Chronic perivascular inoculati ...... s in plaque formation in vivo.

@en

Chronic perivascular inoculati ...... s in plaque formation in vivo.

@nl

prefLabel

Chronic perivascular inoculati ...... s in plaque formation in vivo.

@en

Chronic perivascular inoculati ...... s in plaque formation in vivo.

@nl

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LABINVEST.3700411

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Laboratory Investigation

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Chronic perivascular inoculati ...... s in plaque formation in vivo.

@en

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Constanze Veronika Redl

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Corinna Barz

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Jaroslav Pelisek

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Juergen Heesemann

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Markus Georg Engelmann

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Sigrid Nikol

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P2860

Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections

Chlamydia pneumoniae induces aponecrosis in human aortic smooth muscle cells

Chlamydial and human heat shock protein 60s activate human vascular endothelium, smooth muscle cells, and macrophages.

The atherogenic effects of chlamydia are dependent on serum cholesterol and specific to Chlamydia pneumoniae.

Chlamydia pneumoniae infection promotes a proliferative phenotype in the vasculature through Egr-1 activation in vitro and in vivo.

Chlamydia pneumoniae-induced atherosclerosis in a rabbit model.

Chlamydia pneumoniae antigens, rather than viable bacteria, persist in atherosclerotic lesions.

Chlamydia pneumoniae infections in mouse models: relevance for atherosclerosis research.

Heat-inactivated C. pneumoniae organisms are not atherogenic.

Effect of short-term treatment with azithromycin on recurrent ischaemic events in patients with acute coronary syndrome in the Azithromycin in Acute Coronary Syndrome (AZACS) trial: a randomised controlled trial.

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labinvest.3700411

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467-476

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scholarly article

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10.1038/LABINVEST.3700411

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5

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86

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2006-05-01T00:00:00Z

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7229157

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16550192

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