Chlamydia trachomatis enters a viable but non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells. - Wikidata - awgldk - TriplyDB

Chlamydia trachomatis enters a viable but non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells.

Chlamydia trachomatis enters a viable but non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells.

http://www.wikidata.org/entity/Q37863305

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Severe tryptophan starvation blocks onset of conventional persistence and reduces reactivation of Chlamydia trachomatis The role of peptidoglycan in chlamydial cell division: towards resolving the chlamydial anomaly Water-filtered infrared a irradiation in combination with visible light inhibits acute chlamydial infection Beyond Tryptophan Synthase: Identification of Genes That Contribute to Chlamydia trachomatis Survival during Gamma Interferon-Induced Persistence and Reactivation Chlamydia trachomatis Genital Infections.Chlamydia muridarum enters a viable but non-infectious state in amoxicillin-treated BALB/c mice.Porcine epidemic diarrhea virus (PEDV) co-infection induced chlamydial persistence/stress does not require viral replication.Functional group/guild modelling of inter-specific pathogen interactions: a potential tool for predicting the consequences of co-infection.Chlamydia spp. development is differentially altered by treatment with the LpxC inhibitor LPC-011.Mixed infections with Chlamydia and porcine epidemic diarrhea virus - a new in vitro model of chlamydial persistence Chlamydia trachomatis persistence in vitro: an overview Imbalanced oxidative stress causes chlamydial persistence during non-productive human herpes virus co-infection.Host nectin-1 is required for efficient Chlamydia trachomatis serovar E development.Damage/Danger Associated Molecular Patterns (DAMPs) Modulate Chlamydia pecorum and C. trachomatis Serovar E Inclusion Development In Vitro Chlamydial Pre-Infection Protects from Subsequent Herpes Simplex Virus-2 Challenge in a Murine Vaginal Super-Infection Model.Altered protein secretion of Chlamydia trachomatis in persistently infected human endocervical epithelial cells Progesterone antagonizes the positive influence of estrogen on Chlamydia trachomatis serovar E in an Ishikawa/SHT-290 co-culture model.Host Nectin-1 Promotes Chlamydial Infection in the Female Mouse Genital Tract, but Is Not Required for Infection in a Novel Male Murine Rectal Infection Model Aminomethyl spectinomycins as therapeutics for drug-resistant respiratory tract and sexually transmitted bacterial infections Rottlerin inhibits chlamydial intracellular growth and blocks chlamydial acquisition of sphingolipids from host cells.Penicillin G-Induced Chlamydial Stress Response in a Porcine Strain of Chlamydia pecorum Chlamydia persistence -- a tool to dissect chlamydia--host interactions.What's in a word: the use, misuse, and abuse of the word "persistence" in Chlamydia biology.Commonly prescribed β-lactam antibiotics induce C. trachomatis persistence/stress in culture at physiologically relevant concentrations.STAT3-mediated TLR2/4 pathway upregulation in an IFN-gamma-induced Chlamydia trachomatis persistent infection model.Chlamydia psittaci infection increases mortality of avian influenza virus H9N2 by suppressing host immune response.Chlamydia trachomatis as the Cause of Infectious Infertility: Acute, Repetitive or Persistent Long-Term Infection?Imaging of Chlamydia and host cell metabolism.Autophagy and female genital tract infections: new insights and research directions.The protease inhibitor JO146 demonstrates a critical role for CtHtrA for Chlamydia trachomatis reversion from penicillin persistence.The multifaceted role of oestrogen in enhancing Chlamydia trachomatis infection in polarized human endometrial epithelial cells.Interaction of herpes simplex virus type 2 (HSV-2) glycoprotein D with the host cell surface is sufficient to induce Chlamydia trachomatis persistence.An early event in the herpes simplex virus type-2 replication cycle is sufficient to induce Chlamydia trachomatis persistence.Chlamydia trachomatis: The persistent pathogen.Inhibition of Wnt Signaling Pathways Impairs Chlamydia trachomatis Infection in Endometrial Epithelial Cells.

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Chlamydia trachomatis enters a viable but non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells.

http://www.wikidata.org/entity/Q37863305

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scientific article published on January 2006

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Chlamydia trachomatis enters a ...... HSV-2) co-infected host cells.

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Chlamydia trachomatis enters a viable but non-cultivable

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Chlamydia trachomatis enters a ...... HSV-2) co-infected host cells.

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Chlamydia trachomatis enters a ...... HSV-2) co-infected host cells.

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Chlamydia trachomatis enters a viable but non-cultivable

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J.1462-5822.2005.00608.X

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Cellular Microbiology

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Chlamydia trachomatis enters a ...... HSV-2) co-infected host cells.

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Jennifer Vanover

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Judy Whittimore

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Mary K Howett

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Priscilla B Wyrick

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Robert V Schoborg

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Sophie Dessus-Babus

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Srilekha Deka

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P2860

Sexually Transmitted Diseases Among American Youth: Incidence and Prevalence Estimates, 2000

Persistent chlamydial envelope antigens in antibiotic-exposed infected cells trigger neutrophil chemotaxis

Female to male transmission of human immunodeficiency virus type 1: risk factors for seroconversion in men

Chlamydia pneumoniae expresses genes required for DNA replication but not cytokinesis during persistent infection of HEp-2 cells.

Intracellular survival by Chlamydia.

Chlamydial persistence: beyond the biphasic paradigm.

Differential expression of genes encoding membrane proteins between acute and continuous Chlamydia pneumoniae infections.

Chlamydia trachomatis genes whose products are related to energy metabolism are expressed differentially in active vs. persistent infection.

Herpes simplex virus type 2 modulates the susceptibility of human bladder cells to uropathogenic bacteria.

Expression of Chlamydia trachomatis genes encoding products required for DNA synthesis and cell division during active versus persistent infection.

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149-162

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10.1111/J.1462-5822.2005.00608.X

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1

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8

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2006-01-01T00:00:00Z

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7402747

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16367874

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P921

Chlamydia trachomatis