Silencing or permanent activation: host-cell responses in models of persistent Chlamydia pneumoniae infection.
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Penicillin kills Chlamydia following the fusion of bacteria with lysosomes and prevents genital inflammatory lesions in C. muridarum-infected miceLead Discovery Strategies for Identification of Chlamydia pneumoniae InhibitorsKinematics of intracellular chlamydiae provide evidence for contact-dependent developmentChlamydia pneumoniae hides inside apoptotic neutrophils to silently infect and propagate in macrophages.Chlamydia trachomatis antigens in enteroendocrine cells and macrophages of the small bowel in patients with severe irritable bowel syndrome.Inhibitory effects of 405 nm irradiation on Chlamydia trachomatis growth and characterization of the ensuing inflammatory response in HeLa cellsTranscriptional response patterns of Chlamydophila psittaci in different in vitro models of persistent infection.Simultaneous transcriptional profiling of bacteria and their host cellsChlamydophila pneumoniae phospholipase D (CpPLD) drives Th17 inflammation in human atherosclerosisPenicillin G-Induced Chlamydial Stress Response in a Porcine Strain of Chlamydia pecorumChlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosisPersistently elevated level of IL-8 in Chlamydia trachomatis infected HeLa 229 cells is dependent on intracellular available iron.Interaction of Chlamydiae with human macrophages.Chlamydia pneumoniae infection in atherosclerotic lesion development through oxidative stress: a brief overview.An optimal method of iron starvation of the obligate intracellular pathogen, Chlamydia trachomatis.Host cell responses to Chlamydia pneumoniae in gamma interferon-induced persistence overlap those of productive infection and are linked to genes involved in apoptosis, cell cycle, and metabolism.Sustained interleukin-6 and interleukin-8 expression following infection with Chlamydia trachomatis serovar L2 in a HeLa/THP-1 cell co-culture model.Persistent infection of Chlamydia in reactive arthritis.
P2860
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P2860
Silencing or permanent activation: host-cell responses in models of persistent Chlamydia pneumoniae infection.
description
article científic
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article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on August 2005
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@en
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@nl
type
label
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@en
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@nl
prefLabel
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@en
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@nl
P2093
P2860
P1476
Silencing or permanent activat ...... hlamydia pneumoniae infection.
@en
P2093
Andreas Klos
David Holzberg
Gerda Bartling
Jan Peters
Jessica Thalmann
Katja Endlich
Myriam Schaefer
Simone Hess
P2860
P304
P356
10.1111/J.1462-5822.2005.00534.X
P577
2005-08-01T00:00:00Z