Greater pathogen burden but not elevated C-reactive protein increases the risk of clinical restenosis after percutaneous coronary intervention.
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Neointimal formation is reduced after arterial injury in human crp transgenic miceHelicobacter pylori and extragastric diseases -- other helicobacters.Chlamydia pneumoniae and coronary artery disease: legitimized linkages?Influence of intimal Chlamydophila pneumoniae persistence on cardiovascular complications after coronary intervention.Serum and peripheral blood mononuclear cells infectious burden: correlation to inflammation and atherosclerosis in haemodialysis patients.Pathogen burden, inflammation, proliferation and apoptosis in human in-stent restenosis. Tissue characteristics compared to primary atherosclerosis.
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Greater pathogen burden but not elevated C-reactive protein increases the risk of clinical restenosis after percutaneous coronary intervention.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on September 2002
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Greater pathogen burden but no ...... taneous coronary intervention.
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Greater pathogen burden but no ...... taneous coronary intervention.
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type
label
Greater pathogen burden but no ...... taneous coronary intervention.
@en
Greater pathogen burden but no ...... taneous coronary intervention.
@nl
prefLabel
Greater pathogen burden but no ...... taneous coronary intervention.
@en
Greater pathogen burden but no ...... taneous coronary intervention.
@nl
P2093
P356
P1476
Greater pathogen burden but no ...... taneous coronary intervention.
@en
P2093
Benjamin D Horne
Gunnar G Strobel
Intermountain Heart Collaborative (IHC) Study Group
Jeffrey L Anderson
John F Carlquist
Joseph B Muhlestein
Tami L Bair
P304
P356
10.1067/MHJ.2002.125010
P407
P577
2002-09-01T00:00:00Z