HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
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PPAR Ligands Function as Suppressors That Target Biological Actions of HMGB1Novel concepts on pregnancy clocks and alarms: redundancy and synergy in human parturitionToll-like receptor activation: from renal inflammation to fibrosisImpact of lifetime opioid exposure on arterial stiffness and vascular age: cross-sectional and longitudinal studies in men and women.High mobility group box-1 promotes the proliferation and migration of hepatic stellate cells via TLR4-dependent signal pathways of PI3K/Akt and JNKExpression and significance of HMGB1, TLR4 and NF-κB p65 in human epidermal tumors.Resveratrol ameliorates experimental periodontitis in diabetic mice through negative regulation of TLR4 signalingIntravenous immunoglobulin (IVIg) dampens neuronal toll-like receptor-mediated responses in ischemia.TXNIP links innate host defense mechanisms to oxidative stress and inflammation in retinal Muller glia under chronic hyperglycemia: implications for diabetic retinopathy.Antigen Cross-Priming of Cell-Associated Proteins is Enhanced by Macroautophagy within the Antigen Donor Cell.Toll-like receptor 4 on islet β cells senses expression changes in high-mobility group box 1 and contributes to the initiation of type 1 diabetesHigh mobility group box 1 contributes to the pathogenesis of experimental pulmonary hypertension via activation of Toll-like receptor 4Mucosal immunization with high-mobility group box 1 in chitosan enhances DNA vaccine-induced protection against coxsackievirus B3-induced myocarditis.The Role of HMGB1 in the Pathogenesis of Type 2 Diabetes.Requirement for TLR2 in the development of albuminuria, inflammation and fibrosis in experimental diabetic nephropathy.Ovalbumin modified with pyrraline, a Maillard reaction product, shows enhanced T-cell immunogenicity.Extracellular HMGB1 promotes differentiation of nurse-like cells in chronic lymphocytic leukemia.Serum HMGB1 as a Potential Biomarker for Patients with Asbestos-Related Diseases.Role of GLP-1 and DPP-4 in diabetic nephropathy and cardiovascular disease.The emerging role of the receptor for advanced glycation end products on innate immunity.Role of Toll-like receptors in diabetic nephropathy.Role of high-mobility group box-1 protein in disruption of vascular barriers and regulation of leukocyte-endothelial interactions.HMGB1 enhances the protumoral activities of M2 macrophages by a RAGE-dependent mechanism.Quercetin protects necrotic insult and promotes apoptosis by attenuating the expression of RAGE and its ligand HMGB1 in human breast adenocarcinoma cells.Exogenous high-mobility group box 1 inhibits apoptosis and promotes the proliferation of lewis cells via RAGE/TLR4-dependent signal pathways.Members of the receptor for advanced glycation end products axis as potential therapeutic targets in patients with lupus nephritis.The role of Toll-like receptor proteins (TLR) 2 and 4 in mediating inflammation in proximal tubules.HMGB1: A Promising Therapeutic Target for Prostate Cancer.Lipoxin A4 and its analog suppress inflammation by modulating HMGB1 translocation and expression in psoriasis.Modulation of HMGB1 translocation and RAGE/NFκB cascade by quercetin treatment mitigates atopic dermatitis in NC/Nga transgenic mice.Serum high mobility group box-1 and osteoprotegerin levels are associated with peripheral arterial disease and critical limb ischemia in type 2 diabetic subjects.The dipeptidyl peptidase-4 inhibitor sitagliptin protects against dyslipidemia-related kidney injury in Apolipoprotein E knockout miceUpregulated inflammatory associated factors and blood-retinal barrier changes in the retina of type 2 diabetes mellitus model.Expression of HMGB1 and NF-κB p65 and its significance in non-small cell lung cancer.Effects of high mobility group protein box 1 and toll like receptor 4 pathway on warts caused by human papillomavirus.TLR4-dependant pro-inflammatory effects of HMGB1 on human adipocyte.Association of Toll-like receptor 4 polymorphisms with diabetic foot ulcers and application of artificial neural network in DFU risk assessment in type 2 diabetes patients.Is there a role for high mobility group box 1 and the receptor for advanced glycation end products in the genesis of long-term cognitive impairment in sepsis survivors?TIM-3 as a molecular switch for tumor escape from innate immunity.Anti-septic effects of pellitorine in HMGB1-induced inflammatory responses in vitro and in vivo.
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HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 17 May 2011
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vedecký článok
@sk
vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@en
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@nl
type
label
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@en
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@nl
prefLabel
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@en
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@nl
P2093
P2860
P1476
HMGB1, TLR and RAGE: a functional tripod that leads to diabetic inflammation.
@en
P2093
Caroline Maria de Oliveira Volpe
Clara Araujo Veloso
José Augusto Nogueira-Machado
Miriam Martins Chaves
P2860
P304
P356
10.1517/14728222.2011.575360
P407
P577
2011-05-17T00:00:00Z