Oxidative stress and mitochondrial dysfunction in sepsis.
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Antioxidants that protect mitochondria reduce interleukin-6 and oxidative stress, improve mitochondrial function, and reduce biochemical markers of organ dysfunction in a rat model of acute sepsisFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic ratsFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's diseaseRole of stem cells during diabetic liver injuryMitochondrial Mechanisms in Septic CardiomyopathyEndocannabinoids in neuroendopsychology: multiphasic control of mitochondrial functionTriggers, inhibitors, mechanisms, and significance of eryptosis: the suicidal erythrocyte deathRole of Tyrosine Isomers in Acute and Chronic Diseases Leading to Oxidative Stress - A ReviewSepsis pathophysiology and anesthetic considerationProcainamide Inhibits DNA Methylation and Alleviates Multiple Organ Dysfunction in Rats with Endotoxic ShockMitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stressThe intelligence paradox; will ET get the metabolic syndrome? Lessons from and for Earth.Why do they die? Comparison of selected aspects of organ injury and dysfunction in mice surviving and dying in acute abdominal sepsisThe role of the E2F1 transcription factor in the innate immune response to systemic LPS.Lipopolysaccharide-induced weakness in the preterm diaphragm is associated with mitochondrial electron transport chain dysfunction and oxidative stress.Myocardial dysfunction in sepsis: a large, unsolved puzzleSustained high serum malondialdehyde levels are associated with severity and mortality in septic patients.Energy crisis: the role of oxidative phosphorylation in acute inflammation and sepsisTempol, a superoxide dismutase mimetic agent, ameliorates cisplatin-induced nephrotoxicity through alleviation of mitochondrial dysfunction in miceNeutralization of mitochondrial superoxide by superoxide dismutase 2 promotes bacterial clearance and regulates phagocyte numbers in zebrafish.Importance of Toll-like receptor 2 in mitochondrial dysfunction during polymicrobial sepsisMethylglyoxal as a new biomarker in patients with septic shock: an observational clinical study.The many roads to mitochondrial dysfunction in neuroimmune and neuropsychiatric disordersSeptic Shock in Advanced Age: Transcriptome Analysis Reveals Altered Molecular Signatures in Neutrophil Granulocytes.Total antioxidant capacity is associated with mortality of patients with severe traumatic brain injuryActivation of α2 adrenoceptor attenuates lipopolysaccharide-induced hepatic injuryPoly (ADP-ribose) synthetase inhibitor has a heart protective effect in a rat model of experimental sepsis.The tyrosine kinase inhibitor imatinib prevents lung injury and death after intravenous LPS in mice.Correlation of the oxygen radical activity and antioxidants and severity in critically ill surgical patients - study protocol.Lipopolysaccharide Disrupts Mitochondrial Physiology in Skeletal Muscle via Disparate Effects on Sphingolipid Metabolism.Reactive oxygen species-associated molecular signature predicts survival in patients with sepsis.Protein kinase G oxidation is a major cause of injury during sepsis.Gentisic acid sodium salt, a phenolic compound, is superior to norepinephrine in reversing cardiovascular collapse, hepatic mitochondrial dysfunction and lactic acidemia in Pseudomonas aeruginosa septic shock in dogs.Apigenin protects endothelial cells from lipopolysaccharide (LPS)-induced inflammation by decreasing caspase-3 activation and modulating mitochondrial functionAntioxidants and micronutrient supplementation in trauma patients.Strategies to combat chronic critical illness.Resveratrol restores sirtuin 1 (SIRT1) activity and pyruvate dehydrogenase kinase 1 (PDK1) expression after hemorrhagic injury in a rat model.Inhibition of hepatic cells pyroptosis attenuates CLP-induced acute liver injury.Low paraoxonase 1 activity predicts mortality in surgical patients with sepsis.Cold-inducible RNA-binding protein (CIRP) causes sepsis-associated acute lung injury via induction of endoplasmic reticulum stress.
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Oxidative stress and mitochondrial dysfunction in sepsis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 19 May 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Oxidative stress and mitochondrial dysfunction in sepsis.
@en
Oxidative stress and mitochondrial dysfunction in sepsis.
@nl
type
label
Oxidative stress and mitochondrial dysfunction in sepsis.
@en
Oxidative stress and mitochondrial dysfunction in sepsis.
@nl
prefLabel
Oxidative stress and mitochondrial dysfunction in sepsis.
@en
Oxidative stress and mitochondrial dysfunction in sepsis.
@nl
P356
P1476
Oxidative stress and mitochondrial dysfunction in sepsis.
@en
P2093
H F Galley
P356
10.1093/BJA/AER093
P407
P577
2011-05-19T00:00:00Z