Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Sox-2 Positive Neural Progenitors in the Primate Striatum Undergo Dynamic Changes after Dopamine DenervationDopaminergic-like neurons derived from oral mucosa stem cells by developmental cues improve symptoms in the hemi-parkinsonian rat modelCortex Fraxini (Qingpi) Protects Rat Pheochromocytoma Cells against 6-Hydroxydopamine-Induced ApoptosisIron Deposition Leads to Neuronal α-Synuclein Pathology by Inducing Autophagy Dysfunction.Astrocyte-like cells derived from human oral mucosa stem cells provide neuroprotection in vitro and in vivo.Mesenchymal Stem Cells of Dental Origin-Their Potential for Antiinflammatory and Regenerative Actions in Brain and Gut Damage.Harnessing neurogenesis for the possible treatment of Parkinson's disease.Genetic modification of H2AX renders mesenchymal stromal cell-derived dopamine neurons more resistant to DNA damage and subsequent apoptosis.Dopaminergic cells, derived from a high efficiency differentiation protocol from umbilical cord derived mesenchymal stem cells, alleviate symptoms in a Parkinson's disease rodent model.Liver X receptors agonist promotes differentiation of rat bone marrow derived mesenchymal stem cells into dopaminergic neuron-like cells.
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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article científic
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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P2093
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Cell replacement therapy for Parkinson's disease: how close are we to the clinic?
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Daniel Offen
Eldad Melamed
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10.1586/ERN.11.74
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2011-09-01T00:00:00Z