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NF-κB Pathways in the Pathogenesis of Multiple Sclerosis and the Therapeutic ImplicationsRole of the immunogenic and tolerogenic subsets of dendritic cells in multiple sclerosisNeuroprotection effect of interleukin (IL)-17 secreted by reactive astrocytes is emerged from a high-level IL-17-containing environment during acute neuroinflammationMicroRNA-21 promotes Th17 differentiation and mediates experimental autoimmune encephalomyelitisNarcolepsy and A(H1N1)pdm09 vaccination: shaping the research on the observed signal.Bhlhe40 controls cytokine production by T cells and is essential for pathogenicity in autoimmune neuroinflammation.Biological evaluation of subglutinol a as a novel immunosuppressive agent for inflammation intervention.CNS myelin induces regulatory functions of DC-SIGN-expressing, antigen-presenting cells via cognate interaction with MOGCirculating concentrations of interleukin (IL)-17 in patients with multiple sclerosis: Evaluation of the effects of gender, treatment, disease patterns and IL-23 receptor gene polymorphismsInvolvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.Rescue of notch-1 signaling in antigen-specific CD8+ T cells overcomes tumor-induced T-cell suppression and enhances immunotherapy in cancerPERK-dependent activation of JAK1 and STAT3 contributes to endoplasmic reticulum stress-induced inflammation.Targeting the shift from M1 to M2 macrophages in experimental autoimmune encephalomyelitis mice treated with fasudil.STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation.Synergy between CD8 T cells and Th1 or Th2 polarised CD4 T cells for adoptive immunotherapy of brain tumoursCiglitazone, a peroxisome proliferator-activated receptor gamma ligand, inhibits proliferation and differentiation of th17 cells.Neutrophil-related factors as biomarkers in EAE and MS.Mice deficient in interferon-gamma or interferon-gamma receptor 1 have distinct inflammatory responses to acute viral encephalomyelitis.Effects of Bu Shen Yi Sui Capsule on Th17/Treg cytokines in C57BL/6 mice with experimental autoimmune encephalomyelitisInterleukin-22 is increased in multiple sclerosis patients and targets astrocytesSTAT4 controls GM-CSF production by both Th1 and Th17 cells during EAE.The role of Th cell subsets in the control of Helicobacter infections and in T cell-driven gastric immunopathology.Aging diminishes the resistance of AO rats to EAE: putative role of enhanced generation of GM-CSF Expressing CD4+ T cells in aged rats.Cytokine-neuroantigen fusion proteins as a new class of tolerogenic, therapeutic vaccines for treatment of inflammatory demyelinating disease in rodent models of multiple sclerosisUnique and redundant functions of NKp46+ ILC3s in models of intestinal inflammationGM-CSF-Producing Th Cells in Rats Sensitive and Resistant to Experimental Autoimmune Encephalomyelitis.Elevated expression of interleukin-21 and its correlation to T-cell subpopulation in patients with ulcerative colitis.Prostaglandin E2-induced IL-23p19 subunit is regulated by cAMP-responsive element-binding protein and C/AATT enhancer-binding protein β in bone marrow-derived dendritic cellsEndogenous adaptation to low oxygen modulates T-cell regulatory pathways in EAE.2-Methoxyestradiol inhibits experimental autoimmune encephalomyelitis through suppression of immune cell activationCarbon nanospheres mediated delivery of nuclear matrix protein SMAR1 to direct experimental autoimmune encephalomyelitis in mice.Tbet or Continued RORγt Expression Is Not Required for Th17-Associated Immunopathology.Increased CD8+ T cell responses to apoptotic T cell-associated antigens in multiple sclerosis.The transcription factor Twist1 limits T helper 17 and T follicular helper cell development by repressing the gene encoding the interleukin-6 receptor α chain.Autoantibodies to Non-myelin Antigens as Contributors to the Pathogenesis of Multiple Sclerosis.Diverting T helper cell trafficking through increased plasticity attenuates autoimmune encephalomyelitis.Inhibition of the immunoproteasome ameliorates experimental autoimmune encephalomyelitis.Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.Regulation of experimental autoimmune encephalomyelitis by TPL-2 kinase.Comparison of microglia and infiltrating CD11c⁺ cells as antigen presenting cells for T cell proliferation and cytokine response.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
T
@nl
T(H)17 cytokines in autoimmune neuro-inflammation.
@en
type
label
T
@nl
T(H)17 cytokines in autoimmune neuro-inflammation.
@en
prefLabel
T
@nl
T(H)17 cytokines in autoimmune neuro-inflammation.
@en
P2860
P1476
T(H)17 cytokines in autoimmune neuro-inflammation
@en
P2093
Burkhard Becher
P2860
P304
P356
10.1016/J.COI.2011.08.005
P577
2011-09-09T00:00:00Z