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The Case for the Use of PPARγ Agonists as an Adjunctive Therapy for Cerebral Malaria.PPARγ agonists improve survival and neurocognitive outcomes in experimental cerebral malaria and induce neuroprotective pathways in human malaria.Prevention of JNK phosphorylation as a mechanism for rosiglitazone in neuroprotection after transient cerebral ischemia: activation of dual specificity phosphatase.Pleiotropic role of PPARγ in intracerebral hemorrhage: an intricate system involving Nrf2, RXR, and NF-κB.Neuroprotection by rosiglitazone in transient focal cerebral ischemia might not be mediated by glutamate transporter-1.
P2860
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
Therapeutic Potential of PPARγ Activation in Stroke.
@en
Therapeutic Potential of PPARγ Activation in Stroke.
@nl
type
label
Therapeutic Potential of PPARγ Activation in Stroke.
@en
Therapeutic Potential of PPARγ Activation in Stroke.
@nl
prefLabel
Therapeutic Potential of PPARγ Activation in Stroke.
@en
Therapeutic Potential of PPARγ Activation in Stroke.
@nl
P2860
P356
P1433
P1476
Therapeutic Potential of PPARγ Activation in Stroke.
@en
P2093
Raghu Vemuganti
P2860
P304
P356
10.1155/2008/461981
P577
2008-04-13T00:00:00Z