Repair after acute lung injury: molecular mechanisms and therapeutic opportunities. - Wikidata - awgldk - TriplyDB

Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

http://www.wikidata.org/entity/Q37994893

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Pediatric Acute Respiratory Distress Syndrome: Fibrosis versus Repair A Pathophysiologic Approach to Biomarkers in Acute Respiratory Distress Syndrome Silencing Bruton's tyrosine kinase in alveolar neutrophils protects mice from LPS/immune complex-induced acute lung injury.Pathophysiology of lung injury induced by common bile duct ligation in mice.Mesenchymal stromal cells are more effective than the MSC secretome in diminishing injury and enhancing recovery following ventilator-induced lung injury Agonist binding to β-adrenergic receptors on human airway epithelial cells inhibits migration and wound repair.A Systematic Review of the Role of Dysfunctional Wound Healing in the Pathogenesis and Treatment of Idiopathic Pulmonary Fibrosis.Anti-inflammatory effects of clarithromycin in ventilator-induced lung injury.Network clusters analysis based on protein-protein interaction network constructed in phosgene-induced acute lung injury.Early and late pulmonary effects of nebulized LPS in mice: An acute lung injury model Goats without Prion Protein Display Enhanced Proinflammatory Pulmonary Signaling and Extracellular Matrix Remodeling upon Systemic Lipopolysaccharide Challenge.Phosphorylated E2F1 is stabilized by nuclear USP11 to drive Peg10 gene expression and activate lung epithelial cells.Neutrophils in tissue injury and repair.Mechanical stretch modulates cell migration in the lungs.Host gene expression profiles in ferrets infected with genetically distinct henipavirus strains.Accumulation of Multipotent Progenitor Cells on Polymethylpentene Membranes During Extracorporeal Membrane Oxygenation.Protective Effects of Anti-IL17 on Acute Lung Injury Induced by LPS in Mice

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P2860

Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

http://www.wikidata.org/entity/Q37994893

description

article científic

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article scientifique

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articol științific

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artigo científico

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artikel ilmiah

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artículo científico

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name

Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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type

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Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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prefLabel

Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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Repair after acute lung injury: molecular mechanisms and therapeutic opportunities.

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Repair after acute lung injury: molecular mechanisms and therapeutic opportunities

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Guillermo M Albaiceta

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Randomized, placebo-controlled clinical trial of an aerosolized β₂-agonist for treatment of acute lung injury.

Lung tissue regeneration after induced injury in Runx3 KO mice.

Neutrophil depletion blocks early collagen degradation in repairing cholestatic rat livers

EGFR tyrosine kinase inhibition worsens acute lung injury in mice with repairing airway epithelium.

Resistance to bleomycin-induced lung fibrosis in MMP-8 deficient mice is mediated by interleukin-10

Neutrophils and acute lung injury.

IV delivery of induced pluripotent stem cells attenuates endotoxin-induced acute lung injury in mice.

Chemokine and cytokine processing by matrix metalloproteinases and its effect on leukocyte migration and inflammation.

FoxM1 mediates the progenitor function of type II epithelial cells in repairing alveolar injury induced by Pseudomonas aeruginosa.

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209

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scholarly article

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10.1186/CC11224

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2

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16

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Adrian Gonzalez-Lopez

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2012-12-12T00:00:00Z

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221722320

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1053054996

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22429641

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3681355

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