Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons. - Wikidata - awgldk - TriplyDB

Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

http://www.wikidata.org/entity/Q37998457

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Overexpression of Rad51C splice variants in colorectal tumors Emerging complexity of the HuD/ELAVl4 gene; implications for neuronal development, function, and dysfunction Dioxin and dioxin-like compounds suppress acetylcholinesterase activity via transcriptional downregulations in vitro.Acetylcholinesterase: old questions and new developments.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

http://www.wikidata.org/entity/Q37998457

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article scientifique

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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FNMOL.2012.00036

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Frontiers in molecular neuroscience

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Trans-acting factors governing acetylcholinesterase mRNA metabolism in neurons.

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Bernard J Jasmin

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Lucas M Bronicki

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P2860

Acetylcholinesterase--new roles for an old actor

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The ELAV RNA-stability factor HuR binds the 5'-untranslated region of the human IGF-IR transcript and differentially represses cap-dependent and IRES-mediated translation

Pumilio-2 function in the mouse nervous system

Visualization of translated tau protein in the axons of neuronal P19 cells and characterization of tau RNP granules

Post-transcriptional regulation of acetylcholinesterase mRNAs in nerve growth factor-treated PC12 cells by the RNA-binding protein HuD

RNA-protein interactions and control of mRNA stability in neurons

MicroRNA-132 potentiates cholinergic anti-inflammatory signaling by targeting acetylcholinesterase

Gene structure of mammalian acetylcholinesterase. Alternative exons dictate tissue-specific expression

Virtues and woes of AChE alternative splicing in stress-related neuropathologies

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36

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scholarly article

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10.3389/FNMOL.2012.00036

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2012-03-22T00:00:00Z

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223990494

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