Impairments of synaptic plasticity in aged animals and in animal models of Alzheimer's disease - Wikidata - awgldk - TriplyDB

Impairments of synaptic plasticity in aged animals and in animal models of Alzheimer's disease

Impairments of synaptic plasticity in aged animals and in animal models of Alzheimer's disease

http://www.wikidata.org/entity/Q38005423

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Memory consolidation in aging and MCI after 1 week.Shared genetic etiology underlying Alzheimer's disease and type 2 diabetes.Life-time expression of the proteins peroxiredoxin, beta-synuclein, PARK7/DJ-1, and stathmin in the primary visual and primary somatosensory cortices in rats.Neuropathological approaches to cerebral aging and neuroplasticity.Brain response to calorie restriction.Mechanisms underlying synaptic vulnerability and degeneration in neurodegenerative disease.Tripeptidyl Peptidase II Mediates Levels of Nuclear Phosphorylated ERK1 and ERK2.Differential Roles of Environmental Enrichment in Alzheimer's Type of Neurodegeneration and Physiological Aging.Therapeutic efficacy of the Ginkgo special extract EGb761® within the framework of the mitochondrial cascade hypothesis of Alzheimer's disease.Protective Effects of Proline-Rich Peptide in a Rat Model of Alzheimer Disease: An Electrophysiological Study.Reduced Balance Restoration Capacities Following Unilateral Vestibular Insult in Elderly Mice.

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Impairments of synaptic plasticity in aged animals and in animal models of Alzheimer's disease

http://www.wikidata.org/entity/Q38005423

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Rejuvenation Research

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Impairments of synaptic plasti ...... models of Alzheimer's disease

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Costanza Burattini

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Daniela Platano

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Patrizia Fattoretti

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P2860

Apolipoprotein E4: a causative factor and therapeutic target in neuropathology, including Alzheimer's disease

New insights into brain BDNF function in normal aging and Alzheimer disease

ApoE4 reduces glutamate receptor function and synaptic plasticity by selectively impairing ApoE receptor recycling

AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss

Cysteine regulation of protein function--as exemplified by NMDA-receptor modulation.

Reversal of age-related alterations in synaptic plasticity by blockade of L-type Ca2+ channels.

Amyloid-beta and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline

Hippocampal neurons recycle BDNF for activity-dependent secretion and LTP maintenance.

A ketogenic diet increases succinic dehydrogenase (SDH) activity and recovers age-related decrease in numeric density of SDH-positive mitochondria in cerebellar Purkinje cells of late-adult rats.

Intracellular redox state alters NMDA receptor response during aging through Ca2+/calmodulin-dependent protein kinase II

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235-238

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scholarly article

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10.1089/REJ.2012.1318

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2

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15

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Giorgio Aicardi

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Fabrizia Lattanzio

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2012-04-01T00:00:00Z

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224848248

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22533439

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Alzheimer's disease

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3332390

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