Considerations regarding the etiology and future treatment of autosomal recessive versus idiopathic Parkinson disease. - Wikidata - awgldk - TriplyDB

Considerations regarding the etiology and future treatment of autosomal recessive versus idiopathic Parkinson disease.

Considerations regarding the etiology and future treatment of autosomal recessive versus idiopathic Parkinson disease.

http://www.wikidata.org/entity/Q38006645

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Systems analysis of genetic variation in MPTP neurotoxicity in mice Parkin-dependent degradation of the F-box protein Fbw7β promotes neuronal survival in response to oxidative stress by stabilizing Mcl-1 Modelling idiopathic Parkinson disease as a complex illness can inform incidence rate in healthy adults: the PREDIGT score Holocranohistochemistry enables the visualization of α-synuclein expression in the murine olfactory system and discovery of its systemic anti-microbial effects Dopaminergic neuronal imaging in genetic Parkinson's disease: insights into pathogenesis Lipid profiling of parkin-mutant human skin fibroblasts.Progressive dopaminergic cell loss with unilateral-to-bilateral progression in a genetic model of Parkinson disease Early synaptic dysfunction in Parkinson's disease: Insights from animal models.Outlining a Population "at Risk" of Parkinson's Disease: Evidence from a Case-Control Study.Differential Effects of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine on Motor Behavior and Dopamine Levels at Brain Regions in Three Different Mouse Strains.

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Considerations regarding the etiology and future treatment of autosomal recessive versus idiopathic Parkinson disease.

http://www.wikidata.org/entity/Q38006645

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S11940-012-0175-8

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Current treatment options in neurology

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Considerations regarding the e ...... s idiopathic Parkinson disease

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David A Grimes

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Hei Sio Ao

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Tohru Kitada

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P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase

Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

PINK1 protein in normal human brain and Parkinson's disease

The kinase LRRK2 is a regulator of the transcription factor NFAT that modulates the severity of inflammatory bowel disease

Imputation of sequence variants for identification of genetic risks for Parkinson's disease: a meta-analysis of genome-wide association studies

Multicenter analysis of glucocerebrosidase mutations in Parkinson's disease.

Mutations in LRRK2 cause autosomal-dominant parkinsonism with pleomorphic pathology

Lewy body-like pathology in long-term embryonic nigral transplants in Parkinson's disease

'Rejuvenation' protects neurons in mouse models of Parkinson's disease

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s11940-012-0175-8

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230-240

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scholarly article

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10.1007/S11940-012-0175-8

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3

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14

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Michael G Schlossmacher

Julianna J Tomlinson

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2012-06-01T00:00:00Z

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22547255

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Parkinson's disease