Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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P1476

Antigen presentation in MHC class II transgenic mice: stimulation versus tolerization.

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P2093

Brinster RL

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Burkly LC

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Flavell RA

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Lo D

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Palmiter RD

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P2860

Organ-specific autoimmune diseases induced in mice by elimination of T cell subset. I. Evidence for the active participation of T cells in natural self-tolerance; deficit of a T cell subset as a possible cause of autoimmune disease.

Study on cellular events in post-thymectomy autoimmune oophoritis in mice. II. Requirement of Lyt-1 cells in normal female mice for the prevention of oophoritis.

Self tolerance and localized autoimmunity. Mouse models of autoimmune disease that suggest tissue-specific suppressor T cells are involved in self tolerance.

Antigen presentation by chemically modified splenocytes induces antigen-specific T cell unresponsiveness in vitro and in vivo

Rare peripheral T cells migrate to and persist in normal mouse thymus.

T cell-mediated inhibition of the transfer of autoimmune diabetes in NOD mice.

Tolerance in transgenic mice expressing class II major histocompatibility complex on pancreatic acinar cells.

Antigen-presenting cells for unprimed T cells.

Diabetes and tolerance in transgenic mice expressing class II MHC molecules in pancreatic beta cells.

T cells can distinguish between allogeneic major histocompatibility complex products on different cell types.

P304

121-134

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P31

scholarly article

P356

10.1111/J.1600-065X.1990.TB00570.X

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P478

117

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P577

1990-10-01T00:00:00Z

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P698

2258189

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